Results 161 to 170 of about 8,577,289 (274)
The Hyers-Ulam-Rassias Stability of ( ð ‘š, ð ‘› ) ( 𠜎, ð œ ) -Derivations on Normed Algebras
Ajda Fošner
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Methods to improve antibody–drug conjugate (ADC) treatment durability in cancer therapy are needed. We utilized ADCs and immune‐stimulating antibody conjugates (ISACs), which are made from two non‐competitive antibodies, to enhance the entry of toxic payloads into cancer cells and deliver immunostimulatory agents into immune cells.
Tiexin Wang +3 more
wiley +1 more source
Artificial neural network model of the capacity of diethylenetriamine functionalised zinc oxide-apricot stone shell nanocomposite for sequestering 2,4-dichlorophenoxyacetic acid from wastewater. [PDF]
Akinola AO +3 more
europepmc +1 more source
ÐÐÐÐÐÐÐСÐРСÐСТÐÐÐ ÐÐÐ¥ÐСТУ ÐÐФÐÐ ÐÐЦÐÐ ÐÐÐÐÐЬÐÐРСÐÐÐÐÐÐСТÐ
ÐÑÑенко, Т.Ð.
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Etoposide induces DNA damage, activating p53‐dependent apoptosis via caspase‐3/7, which cleaves PARP1. Dammarenediol II enhances this apoptotic pathway by suppressing O‐GlcNAc transferase activity, further decreasing O‐GlcNAcylation. The reduction in O‐GlcNAc levels boosts p53‐driven apoptosis and influences the Akt/GSK3β/mTOR signaling pathway ...
Jaehoon Lee +8 more
wiley +1 more source
Structural Fiber Tract Alterations in Relation to Surgery in Children With a Posterior Fossa Tumor. [PDF]
Jellema PEJ +7 more
europepmc +1 more source
Tumor mutational burden as a determinant of metastatic dissemination patterns
This study performed a comprehensive analysis of genomic data to elucidate whether metastasis in certain organs share genetic characteristics regardless of cancer type. No robust mutational patterns were identified across different metastatic locations and cancer types.
Eduardo Candeal +4 more
wiley +1 more source
Correction to: Dp44mT targets the AKT, TGF-β and ERK pathways via the metastasis suppressor NDRG1 in normal prostate epithelial cells and prostate cancer cells. [PDF]
Dixon KM +8 more
europepmc +1 more source
Aptamers are used both therapeutically and as targeting agents in cancer treatment. We developed an aptamer‐targeted PLGA–TRAIL nanosystem that exhibited superior therapeutic efficacy in NOD/SCID breast cancer models. This nanosystem represents a novel biotechnological drug candidate for suppressing resistance development in breast cancer.
Gulen Melike Demirbolat +8 more
wiley +1 more source

