[Mechanism of <i>Tongxie Yaofang</i> for reducing 5-HT-induced oxidative stress injury in BRL 3A cells: the mediating role of the SREBP-1/CD1 pathway]. [PDF]
Song M +5 more
europepmc +1 more source
[Ultrafine <i>Xiebai</i> powder alleviates lipopolysaccharide-induced acute lung injury in mice by suppressing ferroptosis and modulating the NF-κB/STAT3 signaling axis]. [PDF]
Zhu J +8 more
europepmc +1 more source
[Hypolipidemic activity of phenolic acids from <i>Inula japonica</i> Thunb.] [PDF]
Chen Y +5 more
europepmc +1 more source
外源性硫化氢通过抗氧化应激逆转过氧化氢诱导的 人脐静脉内皮细胞衰老
摘 要: 【目的】 探讨硫化氢(H2S)对过氧化氢(H2O2)诱导的人脐静脉内皮细胞衰老的影响及其抗氧化机制。【方法】 建立H2O2诱导的人脐静脉内皮细胞衰老模型,采用不同浓度的硫氢化钠(NaHS)预处理内皮细胞,观察其对衰老的作用。【结果】 与对照组相比,60 μmol/L H2O2预处理后,能显著提高细胞SA β-gal阳性率和血浆纤溶酶原激活物抑制剂-1(PAI-1)的表达,同时增加丙二醛(MDA)含量,但减少过氧化物歧化酶1(SOD1)的表达。而NaHS的预处理可以显著减少细胞SA β ...
doaj
[Effects of glutathione on acute cutaneous photodamage in mice and its mechanism]. [PDF]
Wang Q +5 more
europepmc +1 more source
枸杞多糖通过激活Nrf2/HO-1通路保护HK-2细胞氧化损伤的作用 附视频
目的 分析枸杞多糖(LBP)干预对氧化损伤的人肾小管上皮细胞(HK-2)内的核因子E2相关因子2(Nrf2)/血红素氧合酶-1(HO-1)通路蛋白表达的变化,探索LBP拮抗HK-2细胞氧化损伤的分子机制。方法 采用过氧化氢诱导HK-2细胞制备氧化损伤细胞模型,HK-2被分成4组:正常组、LBP组、氧化损伤组及LBP干预组。观察4组细胞的长势和形态变化;细胞活力测定法比较每组细胞的存活率;比色法分析氧化产物丙二醛(malondialdehyde, MDA)含量以及超氧化物歧化酶(superoxide ...
doaj +2 more sources
[Research progress on the mechanism of mitochondrial dynamics in diabetic foot ulcers and its related intervention strategies]. [PDF]
Shan B +6 more
europepmc +1 more source
[A case of neonatal fatty acid oxidation disorder]. [PDF]
Deng LL +4 more
europepmc +1 more source
[Aerobic exercise produces cardioprotective effects in mice by regulating the oxidative stress-inflammation-Hippo/YAP signaling axis]. [PDF]
Zhang G +5 more
europepmc +1 more source
以神东集团柳塔矿区餐厨垃圾为例,研究了过氧化氢(H2O2)预处理对餐厨垃圾性质及其厌氧发酵产酸效能的影响,并考察厌氧发酵液作为矿区污水处理厂碳源的脱氮除磷效果。结果发现,H2O2预处理可有效促进餐厨垃圾中溶解性有机物的释放,同时强化餐厨垃圾浆液的流动性,H2O2预处理后挥发性脂肪酸(VFAs)产量从10 810~18 131 mg/L增大到21 000~28 000 mg/L,发酵产物从丁酸转化为乙酸,餐厨垃圾厌氧发酵酸化效率大幅度提升 ...
车冬妮 +4 more
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