Results 271 to 280 of about 170,290 (345)
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Ventral Abdominal Wall Defects
Pediatrics In Review, 20191. Sara A. Mansfield, MD, MS* 2. Tim Jancelewicz, MD, MA, MS* 1. *Division of Pediatric Surgery, Le Bonheur Children’s Hospital, University of Tennessee Health Science Center, Memphis, TN Clinicians should be aware of the strategies for prenatal and postnatal management of infants with omphalocele and gastroschisis.
Sara A. Mansfield, Tim Jancelewicz
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Pediatric Abdominal Wall Defects
Surgical Clinics of North America, 2013This article reviews the incidence, presentation, anatomy, and surgical management of abdominal wall defects found in the pediatric population. Defects such as inguinal hernia and umbilical hernia are common and are encountered frequently by the pediatric surgeon. Recently developed techniques for repairing these hernias are aimed at improving cosmesis
Todd A. Ponsky, Katherine B. Kelly
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Two main types of abdominal wall defects were studied, omphalocele (n=133) and gastroschisis (n=194). Low maternal age was associated with a low risk for omphalocele but a high risk for gastroschisis. For both types of malformation the risk at parity 1 was higher than at higher parity.
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Neonatal abdominal wall defects
Seminars in Fetal and Neonatal Medicine, 2011Gastroschisis and omphalocele are the two most common congenital abdominal wall defects. Both are frequently detected prenatally due to routine maternal serum screening and fetal ultrasound. Prenatal diagnosis may influence timing, mode and location of delivery.
Emily R. Christison-Lagay+2 more
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Management of Abdominal Wall Defects
Surgical Clinics of North America, 2022Congenital abdominal wall defects vary from abdominal wall hernias to severe congenital structural anomalies that include gastroschisis, omphalocele, and prune belly syndrome. The conditions often carry various associated anomalies and require multidisciplinary treatment approaches. Complex surgical reconstructive techniques are frequently required and
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1998
In this section, two main categories of pathological destruction of abdominal wall tissue will be discussed, i.e., open trauma (burns, shotgun wounds, dilaceration) and infection of traumatic, operative, or apparently spontaneous origin. These lesions raise difficult therapeutic problems regarding the extent of exeresis and the subsequent covering of ...
J. B. Flament+3 more
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In this section, two main categories of pathological destruction of abdominal wall tissue will be discussed, i.e., open trauma (burns, shotgun wounds, dilaceration) and infection of traumatic, operative, or apparently spontaneous origin. These lesions raise difficult therapeutic problems regarding the extent of exeresis and the subsequent covering of ...
J. B. Flament+3 more
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Challenging abdominal wall defects
The American Journal of Surgery, 2001We propose a simple algorithm for management of patients with challenging abdominal fascial defects.The medical records of 64 patients with complicated abdominal wall defects representing a consecutive series by a single surgeon over a 4-year period were reviewed. Group I patients presented with massive fascial defects and closed wounds.
Samir M. Sukkar+3 more
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Familial abdominal wall defects
American Journal of Medical Genetics, 1989AbstractWe report 2 families, each having multiple sibs with abdominal wall defects. In family 1, normal parents gave birth to identical (monochorionic, diamniotic) twins. This is the first reported case of gastroschisis occurring in monozygotic twins. In family 2, a normal mother gave birth to a son with omphalocele.
Hanns C. Haesslein+3 more
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Congenital Abdominal Wall Defects
Clinics in Perinatology, 1978Congenital abdominal wall defects are among the more common anomalies encountered by pediatric surgeons. The author's experience with omphalocele and gastrochisis is presented.
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2010
At about 8 weeks gestation, the enlarging liver causes the displacement of other viscera outside the umbilical ring, to return by 10 weeks. Failure to do this results in exomphalos. Thus, it should be covered with sac and Wharton’s jelly with insertion of the cord at its apex.
Mark Davenport, Chandrasen K. Sinha
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At about 8 weeks gestation, the enlarging liver causes the displacement of other viscera outside the umbilical ring, to return by 10 weeks. Failure to do this results in exomphalos. Thus, it should be covered with sac and Wharton’s jelly with insertion of the cord at its apex.
Mark Davenport, Chandrasen K. Sinha
openaire +2 more sources