Results 161 to 170 of about 19,303 (215)
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Journal of Urology, 1984
Most conditions that affect the kidney cause a proportionate and simultaneous loss of glomerular and tubular function. The loss of glomerular function results in the retention of many products of metabolism, including the anions of various organic and inorganic acids, and urea.
T, Pohlman, K A, Hruska, M, Menon
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Most conditions that affect the kidney cause a proportionate and simultaneous loss of glomerular and tubular function. The loss of glomerular function results in the retention of many products of metabolism, including the anions of various organic and inorganic acids, and urea.
T, Pohlman, K A, Hruska, M, Menon
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Medical Clinics of North America, 1983
The kidney has as one of its major responsibilities the excretion of the daily nonvolatile acid load. On a normal American diet, this amounts to about 1 mEq of nonvolatile acid per kilogram body weight. This acid is excreted in the form of titratable acids (e.g., H2PO4 −) and ammonium (NH4 +).
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The kidney has as one of its major responsibilities the excretion of the daily nonvolatile acid load. On a normal American diet, this amounts to about 1 mEq of nonvolatile acid per kilogram body weight. This acid is excreted in the form of titratable acids (e.g., H2PO4 −) and ammonium (NH4 +).
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Annals of Clinical Biochemistry: International Journal of Laboratory Medicine, 1999
Disorders affecting the overall ability of the renal tubules either to secrete hydrogen ions (H+) or to retain bicarbonate ions (HC03) are known collectively by the term renal tubular acidosis (RTA). The condition was first described clinically in 1935,1 confirmed as a renal tubular disorder in 19462 and designated 'renal tubular acidosis' in 1951.3 It
M D, Penney, D A, Oleesky
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Disorders affecting the overall ability of the renal tubules either to secrete hydrogen ions (H+) or to retain bicarbonate ions (HC03) are known collectively by the term renal tubular acidosis (RTA). The condition was first described clinically in 1935,1 confirmed as a renal tubular disorder in 19462 and designated 'renal tubular acidosis' in 1951.3 It
M D, Penney, D A, Oleesky
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Annual Review of Medicine, 1969
Renal tubular acidosis (RTA) represents a clinical syndrome characterized by a state of renal tubular insufficiency with regard to the reabsorption of bicarbonate, the excretion of net hydrogen ion, or both, and includes a large number of etiologies. In this condition, in contradistinction to the so-called glomerular acidosis, glomerular function is ...
J, Rodriguez-Soriano, C M, Edelmann
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Renal tubular acidosis (RTA) represents a clinical syndrome characterized by a state of renal tubular insufficiency with regard to the reabsorption of bicarbonate, the excretion of net hydrogen ion, or both, and includes a large number of etiologies. In this condition, in contradistinction to the so-called glomerular acidosis, glomerular function is ...
J, Rodriguez-Soriano, C M, Edelmann
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Pediatric Clinics of North America, 2019
Renal tubular acidosis should be suspected in poorly thriving young children with hyperchloremic and hypokalemic normal anion gap metabolic acidosis, with/without syndromic features. Further workup is needed to determine the type of renal tubular acidosis and the presumed etiopathogenesis, which informs treatment choices and prognosis.
Robert Todd, Alexander, Martin, Bitzan
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Renal tubular acidosis should be suspected in poorly thriving young children with hyperchloremic and hypokalemic normal anion gap metabolic acidosis, with/without syndromic features. Further workup is needed to determine the type of renal tubular acidosis and the presumed etiopathogenesis, which informs treatment choices and prognosis.
Robert Todd, Alexander, Martin, Bitzan
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Pseudo-Renal Tubular Acidosis: Conditions Mimicking Renal Tubular Acidosis
Advances in Chronic Kidney Disease, 2018Hyperchloremic metabolic acidosis, particularly renal tubular acidosis, can pose diagnostic challenges. The laboratory phenotype of a low total carbon dioxide content, normal anion gap, and hyperchloremia may be misconstrued as hypobicarbonatemia from renal tubular acidosis.
Junior, Uduman, Jerry, Yee
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Endocrinology and Metabolism Clinics of North America, 1990
Renal tubular acidosis refers to a group of disorders that result from pure tubular damage without concomitant glomerular damage. They could be hereditary (primary) or acquired (secondary to various disease states like sickle cell disease, obstructive uropathy, postrenal transplant, autoimmune disease, or drugs).
M, Rothstein, C, Obialo, K A, Hruska
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Renal tubular acidosis refers to a group of disorders that result from pure tubular damage without concomitant glomerular damage. They could be hereditary (primary) or acquired (secondary to various disease states like sickle cell disease, obstructive uropathy, postrenal transplant, autoimmune disease, or drugs).
M, Rothstein, C, Obialo, K A, Hruska
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Dimensions of Critical Care Nursing, 2010
Renal tubular acidosis is a relatively uncommon clinical syndrome characterized by the inability of the kidney to adequately excrete hydrogen ions, retain adequate bicarbonate, or both. This syndrome can be categorized into 3 separate disorders, each with unique clinical characteristics.
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Renal tubular acidosis is a relatively uncommon clinical syndrome characterized by the inability of the kidney to adequately excrete hydrogen ions, retain adequate bicarbonate, or both. This syndrome can be categorized into 3 separate disorders, each with unique clinical characteristics.
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Pregnancy and Renal Tubular Acidosis
American Journal of Perinatology, 1999Renal tubular acidosis (RTA) is uncommonly encountered in pregnancy. The risk for these women to develop pregnancy-induced hypertension has not been previously described. The renal defect noted in these women, aggravated by the normal hypervolemia of pregnancy, may predispose to hypertension.
T F, Rowe, K, Magee, F G, Cunningham
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Current Opinion in Pediatrics, 2004
Research in the past several years has led to the understanding of numerous genetic mutations that lead to inheritable forms of distal renal tubular acidosis (dRTA). Most of these mutations affect the physiology of the A-intercalated cells of the renal cortical collecting duct.
Julie A, Nicoletta, George J, Schwartz
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Research in the past several years has led to the understanding of numerous genetic mutations that lead to inheritable forms of distal renal tubular acidosis (dRTA). Most of these mutations affect the physiology of the A-intercalated cells of the renal cortical collecting duct.
Julie A, Nicoletta, George J, Schwartz
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