Results 161 to 170 of about 8,923 (197)
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Fiber density in acute and chronic inflammatory demyelinating polyneuropathy
Muscle & Nerve, 1992AbstractThe fiber density in the deltoid, extensor digitorum communis, and first dorsal interosseous muscles was measured using SFEMG in 11 patients with acute and chronic inflammatory demyelinating polyneuropathy. The fiber density was increased in 58% of the muscles studied. The deltoid muscle was the most abnormal of the 3 muscles studied. There was
M, Gantayat, M, Swash, M S, Schwartz
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Acute clinical onset chronic inflammatory demyelinating polyneuropathy in a dog
Muscle and Nerve, 2011AbstractWe report a case of acute‐onset ambulatory paraparesis with electrophysiological abnormalities compatible with axonal and demyelinating lesions in a Rottweiler dog. Although the clinical findings were compatible with acute canine idiopathic polyneuropathy, postmortem investigations revealed a chronic demyelinating polyneuropathy affecting the ...
Jessica Molin +2 more
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Perineurium contributes to axonal damage in acute inflammatory demyelinating polyneuropathy
Neurology, 2000To assess if axonal damage in severe acute inflammatory demyelinating polyneuropathy (AIDP) correlates with the appearance of epiperineurium in nerve trunks.Increase of endoneurial fluid pressure in nerve trunks possessing epiperineurium may be an important mechanism of axonal damage in AIDP.A 79-year-old man had a 2-day history of acroparesthesias and
J, Berciano +5 more
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A Case of Mercury Toxicity Complicated by Acute Inflammatory Demyelinating Polyneuropathy
Journal of Child Neurology, 2018A 13-year-old African American male presented with 2 months of subacute altered mental status, ptosis, areflexia, disordered gait, constipation, weight loss, abdominal and testicular pain, and hyperhidrosis. Initial workup at our facility was unrevealing until elevated serum mercury level was detected.
Stacey, Evans +2 more
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Acute inflammatory demyelinating polyneuropathy (Guillain—Barré syndrome)
Russian Journal of Archive of PathologyGuillain—Barré syndrome is an acute immune-mediated polyradiculoneuropathy, which is the most common cause of acute paralysis and requires intensive therapy in one third of cases. Considering the fact that the disease is rare and characterized by low mortality, morphological changes in it are described rather modestly, based on isolated examples.
V.V. Swistunov, E.I. Dzhoga
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European Journal of Paediatric Neurology
To identify clinical factors and biomarkers that could contribute to early differential diagnosis of acute inflammatory demyelinating polyneuropathy (AIDP) and acute-onset chronic inflammatory demyelinating polyneuropathy (A-CIDP) in the pediatric population, with limited evidence.We conducted an observational retrospective study of children diagnosed ...
Jiannan Ma, Li Jiang
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To identify clinical factors and biomarkers that could contribute to early differential diagnosis of acute inflammatory demyelinating polyneuropathy (AIDP) and acute-onset chronic inflammatory demyelinating polyneuropathy (A-CIDP) in the pediatric population, with limited evidence.We conducted an observational retrospective study of children diagnosed ...
Jiannan Ma, Li Jiang
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Journal of Clinical Neuroscience, 1998
Acute inflammatory demyelinating polyneuropathy (Guillain-Barré syndrome) developed in three patients receiving chemotherapy for acute lymphoblastic leukaemia or the closely related entity lymphoblastic lymphoma, a relationship that has not been previously described.
A, Grigg, B, Tait, S, Davis, L, Kiers
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Acute inflammatory demyelinating polyneuropathy (Guillain-Barré syndrome) developed in three patients receiving chemotherapy for acute lymphoblastic leukaemia or the closely related entity lymphoblastic lymphoma, a relationship that has not been previously described.
A, Grigg, B, Tait, S, Davis, L, Kiers
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A critical review of therapies in acute and chronic inflammatory demyelinating polyneuropathies
Muscle & Nerve, 1987AbstractAcute and chronic inflammatory demyelinating neuropathies are among the most common treatable neuropathies seen by neurologists. Evidence for effective therapy has only recently been provided by randomized or controlled trials. In the Guillain‐Barré syndrome such evidence does not support the use of corticosteroids or immunosuppressive agents ...
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[Autonomic dysfunction in acute inflammatory demyelinating polyneuropathy].
Revista medico-chirurgicala a Societatii de Medici si Naturalisti din Iasi, 2005Acute inflammatory demyelinating polyneuropathy (AIDP) is an autoimmune process that is characterized by progressive weakness, mild sensory changes and autonomic dysfunction. It is a rare disorder, afflicting about 1 person in 100,000. Yet, since the decline in the number of polio cases, it represents the most common cause of acute neuromuscular ...
D, Cuciureanu, Rodica, Prodan
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Immune attack on the schwann cell surface in acute inflammatory demyelinating polyneuropathy
Annals of Neurology, 1996AbstractThe localization, mode of action, and roles of complement in the Guillain‐Barré syndrome have been controversial. We used high‐resolution immunocytochemistry to localize complement activation products in early stages of the acute inflammatory demyelinating polyneuropathy (AIDP) pattern of Guillain‐Barré syndrome.
C E, Hafer-Macko +7 more
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