Junctions of the AML1/MTG8(ETO) fusion are constant in t(8;21) acute myeloid leukemia detected by reverse transcription polymerase chain reaction [PDF]
, 1993 T Kozu +7 more
openalex +1 more source
Harnessing the Biological Responses Induced by Nanomaterials for Enhanced Cancer Therapy
Aggregate, EarlyView.Nanomaterial (NM)‐induced toxicity can be strategically repurposed for cancer therapy. This review summarizes the mechanism by which NMs selectively activate specific cellular processes to regulate cell fate independently. We also discussed how NMs‐induced biological responses can be leveraged as therapeutic strategies for cancer treatment.
Liting Wang +6 more
wiley +1 more source
Recombinant human granulocyte-macrophage colony-stimulating factor in combination with standard induction chemotherapy in de novo acute myeloid leukemia [PDF]
, 1991 Peter Bettelheim +9 more
openalex +1 more source
Acute Myeloid Leukemia Associated Leukocytic Hypophysitis Presenting with Central Diabetes Insipidus
AACE Clinical Case Reports, 2018 : Objective: Central diabetes insipidus is a rare complication of acute myeloid leukemia and myelodysplastic syndrome. To date there are fewer than 100 cases reported in the literature, and no published case reports in the endocrinology literature.
Joshua A. Levine, MD, PhD +2 more
doaj
Autologous bone marrow transplantation for acute myeloid leukemia using busulfan plus etoposide as a preparative regimen [PDF]
, 1993 CA Linker +4 more
openalex +1 more source
Defective c-myc and c-myb RNA turnover in acute myeloid leukemia cells [PDF]
, 1992 MR Baer, P Augustinos, AJ Kinniburgh
openalex +1 more source
Involvement of tumor necrosis factor (TNF) receptors p55 and p75 in TNF responses of acute myeloid leukemia blasts in vitro [PDF]
, 1992 R Delwel +3 more
openalex +1 more source
Therapeutic targeting of myeloid cells in liver fibrosis: Mechanisms and clinical prospects
Animal Models and Experimental Medicine, EarlyView.This review systematically summarizes the origin, recruitment, and differentiation regulation mechanisms of myeloid cells in driving hepatic fibrosis progression. By targeting these three critical phases, potential therapeutic strategies for liver fibrosis can be explored.
Yue Wang +5 more
wiley +1 more source