Results 261 to 270 of about 107,794 (297)
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Current Opinion in Pediatrics, 1997
The etiology of acute pancreatitis in children is widely varied and includes idiopathic, drug-related, congenital, and posttraumatic causes. Most children have abdominal pain and tenderness without evidence of peritonitis, and most patients will have elevated serum amylase levels initially or after a delay of about 12 hours.
J I, Greenfeld, C M, Harmon
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The etiology of acute pancreatitis in children is widely varied and includes idiopathic, drug-related, congenital, and posttraumatic causes. Most children have abdominal pain and tenderness without evidence of peritonitis, and most patients will have elevated serum amylase levels initially or after a delay of about 12 hours.
J I, Greenfeld, C M, Harmon
openaire +2 more sources
Hospital Medicine, 2000
Acute pancreatitis is a common disease. As knowledge of its pathophysiology improves, evidence is found to confirm and refute present management and also to suggest new approaches. This article addresses some of these areas in the context of the management of acute pancreatitis.
M T, Cartmell, A N, Kingsnorth
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Acute pancreatitis is a common disease. As knowledge of its pathophysiology improves, evidence is found to confirm and refute present management and also to suggest new approaches. This article addresses some of these areas in the context of the management of acute pancreatitis.
M T, Cartmell, A N, Kingsnorth
openaire +4 more sources
Current Opinion in Gastroenterology, 1999
Efforts to unravel the intracellular processes that occur in acute pancreatitis continue. In cerulein pancreatitis, new evidence supports the idea that a very early event is premature trypsinogen activation triggered by lysosomal cathepsin B. Clinicians persist in trying to identify more sensitive and specific prognostic signs of the severity of ...
H G, Hotz, H A, Reber
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Efforts to unravel the intracellular processes that occur in acute pancreatitis continue. In cerulein pancreatitis, new evidence supports the idea that a very early event is premature trypsinogen activation triggered by lysosomal cathepsin B. Clinicians persist in trying to identify more sensitive and specific prognostic signs of the severity of ...
H G, Hotz, H A, Reber
openaire +2 more sources
Current Opinion in Gastroenterology, 2005
This review presents advances in our understanding of the pathobiologic responses that mediate acute pancreatitis with an emphasis on the interrelationship between the events occurring in the pancreatic acinar cell and the vascular, neural, and immune systems; information on recent reports describing clinical diagnostic and therapeutic aspects of ...
Dejong, C.H.C. +2 more
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This review presents advances in our understanding of the pathobiologic responses that mediate acute pancreatitis with an emphasis on the interrelationship between the events occurring in the pancreatic acinar cell and the vascular, neural, and immune systems; information on recent reports describing clinical diagnostic and therapeutic aspects of ...
Dejong, C.H.C. +2 more
openaire +5 more sources
Surgical Clinics of North America, 1988
In order to recognize acute pancreatitis in the setting of the acute abdomen, the surgeon must be thoroughly familiar with the numerous etiologies of the disease. No specific test is available to diagnose acute pancreatitis. CT scanning is arguably the most useful single tool, but surgical judgment is critical.
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In order to recognize acute pancreatitis in the setting of the acute abdomen, the surgeon must be thoroughly familiar with the numerous etiologies of the disease. No specific test is available to diagnose acute pancreatitis. CT scanning is arguably the most useful single tool, but surgical judgment is critical.
openaire +2 more sources
Current Opinion in Gastroenterology, 2001
The pathophysiologic concept of acute pancreatitis focuses on early events inside acinar cells such as activation of trypsin. Despite significant progress in the understanding of molecular events in experimental pancreatitis, knowledge of these mechanisms has not yet been translated into therapeutic strategies useful in humans.
Christoph K, Weber, Guido, Adler
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The pathophysiologic concept of acute pancreatitis focuses on early events inside acinar cells such as activation of trypsin. Despite significant progress in the understanding of molecular events in experimental pancreatitis, knowledge of these mechanisms has not yet been translated into therapeutic strategies useful in humans.
Christoph K, Weber, Guido, Adler
openaire +3 more sources