Results 311 to 320 of about 211,518 (344)
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Current Opinion in Gastroenterology, 1999
Efforts to unravel the intracellular processes that occur in acute pancreatitis continue. In cerulein pancreatitis, new evidence supports the idea that a very early event is premature trypsinogen activation triggered by lysosomal cathepsin B. Clinicians persist in trying to identify more sensitive and specific prognostic signs of the severity of ...
H G, Hotz, H A, Reber
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Efforts to unravel the intracellular processes that occur in acute pancreatitis continue. In cerulein pancreatitis, new evidence supports the idea that a very early event is premature trypsinogen activation triggered by lysosomal cathepsin B. Clinicians persist in trying to identify more sensitive and specific prognostic signs of the severity of ...
H G, Hotz, H A, Reber
openaire +2 more sources
Surgical Clinics of North America, 1988
In order to recognize acute pancreatitis in the setting of the acute abdomen, the surgeon must be thoroughly familiar with the numerous etiologies of the disease. No specific test is available to diagnose acute pancreatitis. CT scanning is arguably the most useful single tool, but surgical judgment is critical.
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In order to recognize acute pancreatitis in the setting of the acute abdomen, the surgeon must be thoroughly familiar with the numerous etiologies of the disease. No specific test is available to diagnose acute pancreatitis. CT scanning is arguably the most useful single tool, but surgical judgment is critical.
openaire +2 more sources
Current Opinion in Gastroenterology, 2001
The pathophysiologic concept of acute pancreatitis focuses on early events inside acinar cells such as activation of trypsin. Despite significant progress in the understanding of molecular events in experimental pancreatitis, knowledge of these mechanisms has not yet been translated into therapeutic strategies useful in humans.
Christoph K, Weber, Guido, Adler
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The pathophysiologic concept of acute pancreatitis focuses on early events inside acinar cells such as activation of trypsin. Despite significant progress in the understanding of molecular events in experimental pancreatitis, knowledge of these mechanisms has not yet been translated into therapeutic strategies useful in humans.
Christoph K, Weber, Guido, Adler
openaire +3 more sources
Current Opinion in Gastroenterology, 2005
This review presents advances in our understanding of the pathobiologic responses that mediate acute pancreatitis with an emphasis on the interrelationship between the events occurring in the pancreatic acinar cell and the vascular, neural, and immune systems; information on recent reports describing clinical diagnostic and therapeutic aspects of ...
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This review presents advances in our understanding of the pathobiologic responses that mediate acute pancreatitis with an emphasis on the interrelationship between the events occurring in the pancreatic acinar cell and the vascular, neural, and immune systems; information on recent reports describing clinical diagnostic and therapeutic aspects of ...
openaire +3 more sources
Seminars in Diagnostic Pathology, 2004
Acute pancreatitis is characterized by the occurrence of necroinflammatory changes in the pancreas. Three types of necrosis may be distinguished: (1) interstitial tissue necrosis, which subsequently may also involve acinar and ductal cells, (2) ductal necrosis, and (3) acinar necrosis.
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Acute pancreatitis is characterized by the occurrence of necroinflammatory changes in the pancreas. Three types of necrosis may be distinguished: (1) interstitial tissue necrosis, which subsequently may also involve acinar and ductal cells, (2) ductal necrosis, and (3) acinar necrosis.
openaire +2 more sources

