Results 201 to 210 of about 19,394 (252)
Herpesviruses at Human Aggressive Periodontitis Sites*
M. Radvar, H.R. Arab .
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Comparison of Subgingival and Buccal Mucosa Microbiome in Chronic and Aggressive Periodontitis: A Pilot Study. [PDF]
Wei Y +6 more
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Multidisciplinary Clinical Management of a Localized Aggressive Periodontitis diagnosed in a Child with Glanzmann's Thrombasthenia. [PDF]
Prud'homme T +5 more
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Higher interleukin-33 levels in aggressive periodontitis cases.
Ballambettu SP +3 more
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Arresting aggressive periodontitis
Dental Nursing, 2022Margaret Black outlines aggressive periodontitis and explains how it can be avoided
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Aggressive periodontitis and NOD2 variants
Journal of Human Genetics, 2020Aggressive periodontitis (AgP) occurs at an early age and causes rapid periodontal tissue destruction. Nucleotide-binding oligomerization domain-containing protein 2 (NOD2) encodes a protein with two caspase recruitment domains and eleven leucine-rich repeats.
Noriyoshi Mizuno +17 more
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Microbiology of aggressive periodontitis
Periodontology 2000, 2014AbstractFor decades, Aggregatibacter actinomycetemcomitans has been considered the most likely etiologic agent in aggressive periodontitis. Implementation of DNA‐based microbiologic methodologies has considerably improved our understanding of the composition of subgingival biofilms, and advanced open‐ended molecular techniques even allow for genome ...
Eija K\303\266n\303\266nen +1 more
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Aggressive periodontitis: The unsolved mystery.
Quintessence international (Berlin, Germany : 1985), 2018Aggressive periodontal disease is an oral health mystery. Our current understanding of this disease is that specific bacteria invade the oral cavity and the host reacts with an inflammatory response leading to mass destruction of the alveolar bone. Aggressive periodontal disease is typically observed in a population under the age of 30 and occurs so ...
Danielle, Clark +2 more
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Host response in aggressive periodontitis
Periodontology 2000, 2014AbstractIt is critical to understand the underlying host responses in aggressive periodontitis to provide a better appreciation of the risk and susceptibility to this disease. Such knowledge may elucidate the etiology and susceptibility to aggressive periodontitis and directly influence treatment decisions and aid diagnosis.
Cyelee, Kulkarni, Denis F, Kinane
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