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The role of IL‐12 and TNF‐α in AIDP and AMAN

European Journal of Neurology, 2008
Background:  Acute inflammatory demyelinating polyradiculoneuropathy (AIDP) and acute motor axonal neuropathy (AMAN) have been described as two major subtypes of Guillain‐Barré syndrome (GBS); however, the possible difference of their immune‐inflammatory pathogenesis remains unclear.Methods:  In this study, by using FACS and enzyme‐linked immunosorbent
Tao Jin, A Adem, Bengt Winblad
exaly   +2 more sources

Third AIDP Symposium for Young Penalists

2014
The Third AIDP YP Symposium was dedicated to corporate criminal liability from an international and problem oriented perspective. This chapter contains the minutes of the Symposium as redacted by Stijn Lamberigts.
exaly   +3 more sources

Serial electrophysiological findings in Guillain–Barré syndrome not fulfilling AIDP or AMAN criteria

Journal of Neurology, 2016
Guillain-Barré syndrome (GBS) is categorized into two major subtypes: acute inflammatory demyelinating polyneuropathy (AIDP) and acute motor axonal neuropathy (AMAN). However, a proportion of patients are electrophysiologically unclassified because of electrophysiological findings that do not fulfil AIDP or AMAN criteria, and underlying ...
Takafumi Hosokawa, Fumiharu Kimura
exaly   +3 more sources

AIDP and CIDP having specific antibodies to the carbohydrate epitope (–NeuAcα2–8NeuAcα2–3Galβ1–4Glc–) of gangliosides

Journal of the Neurological Sciences, 2005
Anti-ganglioside antibodies were investigated in plasma exchange solutions (PEs) from two patients with acute and chronic inflammatory demyelinating neuropathies (AIDP and CIDP). Both cases show markedly elevated antibody titers against the lacto-series gangliosides, GM3, GD3, and GT3.
Seigo Usuki   +2 more
exaly   +3 more sources

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