Results 181 to 190 of about 49,507 (253)
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Blood Reviews, 1988
A moderate reduction of plasma antithrombin activity is an uncommon but clinically important cause of severe thromboembolic disease. In recent years the molecule responsible for the major part of this activity (antithrombin III) has been extensively characterised and the mode of inheritance of familial deficiencies worked out.
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A moderate reduction of plasma antithrombin activity is an uncommon but clinically important cause of severe thromboembolic disease. In recent years the molecule responsible for the major part of this activity (antithrombin III) has been extensively characterised and the mode of inheritance of familial deficiencies worked out.
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Neonatal antithrombin III deficiency
The American Journal of Medicine, 1989Hemorrhagic and thrombotic complications are common in sick preterm infants and may reflect inadequate regulation of coagulation. All neonates have low levels of the pivotal regulator antithrombin III (ATIII) compared with adults. Plasma levels of ATIII are very low in preterm infants and are further diminished in infants with respiratory distress ...
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Pathophysiology of Antithrombin III Deficiency
Veterinary Clinics of North America: Small Animal Practice, 1988This article focuses on the pathophysiology of thrombosis in patients with acquired antithrombin III deficiency. Antithrombin III is an important natural inhibitor of the hemostatic mechanism, and a hypercoagulable state is often induced in diseases causing antithrombin III deficiency.
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Hereditary antithrombin III deficiency. Effect of antithrombin III deficiency on platelet function.
The American journal of medicine, 1976Antithrombin III (AT III) is the main physiologic inhibitor of thrombin, and activated factors X and IX as well. Normal levels of AT III appear to be necessary to maintain blood fluidity and to prevent thrombosis. Four families with AT III deficiency and recurrent venous thromboembolism have been reported on. We present an additional family with AT III
A, Carvalho, L, Ellman
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Inherited antithrombin III deficiency and cerebral thrombosis in a child.
Pediatrics, 1980Identification of a family affected by antithrombin III-heparin cofactor (AT-III) deficiency was made after diagnosis of the index case, a 15-year-old boy who suffered cerebral thrombosis.
D. Ambruso, L. Jacobson, W. Hathaway
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Journal of the American Veterinary Medical Association, 1982
The hypercoagulable state accompanying nephrotic syndrome in 3 dogs was characterized by markedly increased fibrinogen and factor VIII concentrations. Antithrombin III was mildly reduced in 1 dog (74% of normal) and markedly reduced in the other 2 dogs (
Green Ra, Kabel Al
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The hypercoagulable state accompanying nephrotic syndrome in 3 dogs was characterized by markedly increased fibrinogen and factor VIII concentrations. Antithrombin III was mildly reduced in 1 dog (74% of normal) and markedly reduced in the other 2 dogs (
Green Ra, Kabel Al
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Romanian journal of internal medicine = Revue roumaine de medecine interne, 1994
Data on clinical features and laboratory diagnosis of familial antithrombin deficiency, a rather heterogeneous group of disorders, are illustrated by observations on two Romanian kindreds afflicted by recurrent thrombotic episodes. In a first family, both plasma antithrombin III antigen and activity were reduced to 50% of normal, a condition ...
M, Cucuianu +6 more
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Data on clinical features and laboratory diagnosis of familial antithrombin deficiency, a rather heterogeneous group of disorders, are illustrated by observations on two Romanian kindreds afflicted by recurrent thrombotic episodes. In a first family, both plasma antithrombin III antigen and activity were reduced to 50% of normal, a condition ...
M, Cucuianu +6 more
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Aortic Thrombosis in Antithrombin III Deficiency
JAMA: The Journal of the American Medical Association, 1982To the Editor.— Although we are told in the article by Michael E. Shapiro, MD, and Edwin W. Salzman, MD (1981;245:1759), that abnormal antithrombin III (AT III) levels were documented in both cases weeks to months after any short-term event or surgery, in case 1 the reduced AT III level was found at a time when resting pain had been present for three ...
J H, Winter, B, Bennett, A S, Douglas
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Antithrombin III deficiency in ischaemic stroke
Klinische Wochenschrift, 1983AT III activity and concentration were measured in 36 patients (mean age 65.5 yrs, range 43-77 yrs) with ischaemic stroke within maximally 48 h of the acute event. In 12 patients (= 33%) AT III activity was reduced below 18.4 IU/ml: 50% of these patients showed normal and 50% reduced AT III concentration of less than 22 mg/dl.
V, Hossmann, W D, Heiss, H, Bewermeyer
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The pregnant antithrombin III deficient patient: Management without antithrombin III concentrate
Thrombosis Research, 1992Pregnant patients with antithrombin III (AT III) deficiency have an unacceptably high risk of venous thromboembolism (VTE). Antithrombotic therapy is therefore recommended. The reported clinical experience of such prophylaxis is limited. Some authors have recommended the use of AT III concentrate in addition to heparin in the management of these ...
E, Blondel-Hill, M J, Mant
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