Results 231 to 240 of about 152,646 (312)
Commercial vascular grafts are made from ePTFE, a highly hydrophobic, foreign material that fails at a high rate in small‐diameter applications. Plasma polymer nanoparticles (PPN) are a versatile material functionalisation tool, used here to present fibrillin‐1 fragment PF8 on the graft surface.
Bob S. L. Lee +9 more
wiley +1 more source
Design, benefits, and use of a hybrid aortic arch stent graft. [PDF]
El-Andari R, Moon MC.
europepmc +1 more source
Cardiac stalling: Modelling energy balance in aortic stenosis and its role in syncope
The Journal of Physiology, EarlyView.
DeWayne Townsend
wiley +1 more source
Gli1+ adventitial stem cells (ASCs) have been thought to generate smooth muscle cells (SMCs) in atherosclerosis. Using a dual‐recombinase lineage tracing to exclude ectopic labeling, Wang et al. found that Gli1+ ASCs do not contribute to SMCs in atherosclerotic plaques.
Haixiao Wang +11 more
wiley +1 more source
Clinical prediction of aortic arch anatomy to guide stroke CT angiography protocols: A machine learning study. [PDF]
Marquez-Romero JM +4 more
europepmc +1 more source
Hafnium (Hf)‐rich carbon dots (Hf‐rCDs) with high metal content (40.7%) and robust batch production capacity (>2 g per batch) are synthesized via a facile air‐assisted pyrolysis method. In vivo CT imaging demonstrates outstanding performance of Hf‐rCDs across multiple physiological systems and enables high‐resolution visualization of swine cervical ...
Shuo Li +11 more
wiley +1 more source
Compression of the Left Brachiocephalic Vein by a Type II Right Aortic Arch: A Rare Vascular Anomaly With Unique Clinical Presentation. [PDF]
Hamade A +4 more
europepmc +1 more source
TRIM40 Drives Pathological Cardiac Hypertrophy and Heart Failure via Ubiquitination of PKN2
This study identifies the E3 ligase TRIM40 as a key driver of pathological cardiac hypertrophy. TRIM40 binds PKN2 via its B‐box domain and, through its C29‐dependent catalytic activity, mediates K63‐linked ubiquitination of PKN2. This modification enhances PKN2 phosphorylation at Ser815, thereby driving hypertrophy.
Risheng Zhao +12 more
wiley +1 more source

