Results 71 to 80 of about 142,868 (338)

Leucine‐Dependent SLC7A5–PGAM5 Interaction Promotes Advanced Atherosclerosis Through Hindering Mitochondrial Function of Macrophages

open access: yesAdvanced Science, EarlyView.
Higher plasma leucine is associated with increased risk of new‐onset myocardial infarction. Leucine deprivation alleviates advanced atherosclerosis in mice. Tumor‐induced leucine deprivation reprograms macrophage metabolism and increases CD5Lhi macrophages in mouse plaques. Mechanistically, leucine deficiency reduced SLC7A5‐PGAM5 binding in macrophages,
Shan Zhong   +22 more
wiley   +1 more source

INFLUENCE OF CARDIOVASCULAR RISK FACTORS ON AORTIC WALL MOTION AFTER REPAIR OF TYPE A AORTIC DISSECTION: AN ECG-GATED CT STUDY [PDF]

open access: yes, 2012
OBJECTIVES: To evaluate aortic shape changes during cardiac cycle with dynamic computed tomographic angiography at important thoracic aorta anatomic landmarks in patients who previously underwent ascending aorta repair because of type A dissection, and ...
CROCCIA, MARIAGRAZIA
core  

Open repair of chronic thoracic and thoracoabdominal aortic dissection using deep hypothermia and circulatory arrest [PDF]

open access: yes, 2017
Background Chronic dissection of the thoracic and thoracoabdominal aorta as sequela of a prior type A or B dissection is a challenging problem that requires close radiographic surveillance and prompt operative intervention in the presence of symptoms ...
Blitzer, David   +6 more
core   +1 more source

Type A aortic dissection secondary to a left common carotid artery dissection

open access: yesRadiology Case Reports, 2019
Common carotid artery dissection is an unusual clinical event that most commonly occurs secondary to type A aortic dissection. We present a rare case of spontaneous common carotid artery dissection temporally preceding aortic dissection.
Simon Doran, MB Bch BAO, MRCPI   +4 more
doaj   +1 more source

Macrophagic Sclerostin Loop2‐ApoER2 Interaction Required by Sclerostin for Cardiovascular Protective Action

open access: yesAdvanced Science, EarlyView.
Sclerostin loop2‐ApoER2 interaction in macrophages is required by sclerostin to suppress NF‐κB nuclear translocation and phosphorylation, to promote macrophage conversion into anti‐inflammatory subtypes in atherosclerotic aortas, as well as to prevent atherosclerosis and aortic aneurysm development in ApoE−/− mice. Abstract Therapeutic antibody against
Luyao Wang   +27 more
wiley   +1 more source

Research progress on the follow⁃up of aortic dissection patients

open access: yesHuli yanjiu, 2020
According to the epidemiology,high⁃risk factors and guideline requirements of aortic dissection,this paper reviewed the follow⁃up information of patients with aortic dissection,such as medication adherence,blood pressure monitoring,exercise and so on.It
LI Miqi, HUANG Sufang
doaj  

Aortic Dissection in a Pregnant Patient without Other Risk Factors

open access: yesCase Reports in Obstetrics and Gynecology, 2019
Background. An aortic dissection is a life-threatening condition in which the intima of the artery tears causing separation of the intima and media.
Priya V. Patel   +3 more
doaj   +1 more source

Acute Type B Aortic Dissection One Month After Fenestrated EVAR Procedure

open access: yesEJVES Short Reports, 2019
Introduction: Acute aortic dissection after endovascular repair of an aortic aneurysm is a rare but serious condition, with potential complications that can result in the death of the patient.
Elias Jose Rodriguez Czaplicki   +3 more
doaj   +1 more source

Acute Type A Aortic Dissection Missed as Acute Coronary Syndrome [PDF]

open access: yesJournal of Clinical and Diagnostic Research, 2016
Although the aortic dissection is not common, its outcome is frequently fatal, and many patients with aortic dissection die before referral to the hospital or any diagnostic testing.
Mohammad Mostafa Ansari-Ramandi   +2 more
doaj   +1 more source

From the Gut to the Brain: Microplastic‐Associated Neurovascular Dysfunction and Implications for Stroke Risk

open access: yesAdvanced Science, EarlyView.
Chronic oral exposure to microplastics may disrupt gut microbiota homeostasis and intestinal barrier integrity, potentially engaging the gut–brain axis and systemic inflammatory responses. These alterations may be associated with impaired blood–brain barrier function, cerebral microvascular dysfunction, and enhanced endothelial inflammation, pro ...
Hongxing Wang   +5 more
wiley   +1 more source

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