Results 161 to 170 of about 296,180 (402)
The Impact of Frailty on Patient-Centered Outcomes Following Aortic Valve Replacement [PDF]
Brian R. Kotajarvi +8 more
openalex +1 more source
Aortic valve replacement in younger patients [PDF]
Richard Whitlock +2 more
openalex +1 more source
Abstract Aims The extent of irreversible cardiomyocyte necrosis after acute myocardial infarction (AMI) is a major determinant of residual left ventricular (LV) function and clinical outcome. Cell therapy based on CD34+ cells has emerged as an option to help repair the myocardium and to improve outcomes.
Jerome Roncalli +17 more
wiley +1 more source
107 Left ventricular intramural haematoma secondary to transcatheter aortic valve replacement [PDF]
Simone Fezzi +7 more
openalex +1 more source
The 366 patients diagnosed with transthyretin amyloidosis cardiomyopathy (ATTR‐CM) were analyzed regarding their clinical characteristics in the first year after approval of tafamidis 61 mg for ATTR‐CM in Germany. Nearly two‐thirds of the patients were in an advanced disease stage and 64% met the key criteria of the “Transthyretin Amyloidosis ...
Richard J. Nies +23 more
wiley +1 more source
Transcatheter Aortic Valve Implantation: Insights into Clinical Complications [PDF]
__Abstract__ Transcatheter Aortic Valve Implantation (TAVI) has emerged as a viable and safe treatment for patients with severe aortic stenosis (AS) who are considered ineligible or at prohibitive risk for Surgical Aortic Valve Replacement (SAVR)1–4.
Boon, R.M.A. (Robert) van der
core +1 more source
Cardiac interventions in patients with achondroplasia: a systematic review. [PDF]
Patients with achondroplasia and other causes of dwarfism suffer from increased rates of cardiovascular disease relative to the remainder of the population.
Choi, Jae Hwan +6 more
core +1 more source
Hypertension in aortic valve disease and its response to valve replacement [PDF]
Alexander V. Zezulka +2 more
openalex +1 more source
Abstract Background and aims The heart is a metabolic organ rich in mitochondria. The failing heart reprograms to utilize different energy substrates, which increase its oxygen consumption. These adaptive changes contribute to increased oxidative stress.
Qinghong Li +12 more
wiley +1 more source

