Results 151 to 160 of about 26,804,949 (344)

Unraveling the epigenetic code in cancer cell–tumor microenvironment crosstalk

open access: yesMolecular Oncology, EarlyView.
Epigenetic regulation is a key driver of cancer development and progression. Diverse epigenetic alterations in cancer cells and components of the tumor microenvironment (TME) orchestrate their communication through multiple mechanisms. We discuss how the epigenetic code coordinates bidirectional cancer cell–TME crosstalk to promote cancer progression ...
Ji Hoon Park, Mi‐Young Kim
wiley   +1 more source

Report (U.S. Atomic Energy Commission)

open access: yes, 1960
From introduction: "This report provides the preliminary design for the nuclear portion of a 300 MWe Mixed Spectrum Superheater to be in operation by ...
U.S. Atomic Energy Commission
core  

Report (U.S. Atomic Energy Commission) [PDF]

open access: yes, 1961
From introduction: "This report presents a conceptual design study of the once-through superheat reactor design based on experimental heat transfer results obtained during the fall of 1960 on Task F-2 of the AEC sponsored Nuclear Superheat Project.
U.S. Atomic Energy Commission
core  

Arginine methylation as a regulatory ratchet in cancer: From substrate selection to malignant‐state stabilization

open access: yesMolecular Oncology, EarlyView.
Arginine methylation can be viewed as a persistence‐prone post‐translational modification regulated by a network of PRMTs. Competitive and compensatory interactions among PRMTs can redistribute methylation across substrate pools shaped by sequence, structural, spatial, and environmental layers, reinforcing RNA‐processing, chromatin, and signaling ...
So Hyun Kwon, Ji Min Lee
wiley   +1 more source

Atomic Energy Commission Report 147

open access: yes, 1966
From introduction: This is one of a series of three reports on airborne radioactivity surveys in the United States. The reports contain the 185 airborne anomaly maps issued by the U. S.
U.S. Atomic Energy Commission. Grand Junction Office.
core  

The VHL tumor suppressor at the crossroad of protein folding, aggregation, and cancer

open access: yesMolecular Oncology, EarlyView.
Mutations, environmental stress, and chaperone dysfunction can destabilize pVHL, promoting its conversion from the native folded state into amyloid‐like assemblies. This transition may contribute to protein storage, cell dormancy, survival, and drug resistance.
Lara Abad   +2 more
wiley   +1 more source

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