Results 121 to 130 of about 5,685 (176)

Pathogenicity of Avian Leukosis Viruses

Avian Diseases, 1982
Three methods were used in attempts to obtain non-oncogenic avian leukosis virus for possible use as an immunoprophylactic agent for the control of lymphoid leukosis in chickens. These were: 1) isolate a nononcogenic virus from commercial breeder flocks experiencing very little or no lymphoid leukosis; 2) obtain a non-oncogenic recombinant from mixed ...
W, Okazaki, H G, Purchase, L B, Crittenden   +2 more
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Effects of Irradiation on Avian Leukosis

American Journal of Veterinary Research, 1971
SUMMARY Whole body x-irradiation did not increase or decrease the frequency of death due to leukosis in chickens inoculated at 22 or 23 days of age or in ovo with rpl-12 virus. The levels of viremia in chickens inoculated in ovo were not increased by irradiation. Whole body irradiation (500 R) did not affect the frequency, progression, or regression of
R W, Loan, M M, Storm, K C, Sloan, E L, McCune   +3 more
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Avian Leukosis: Historical Perspectives

Avian Diseases, 1975
What is a perspective of history and how might it be useful today? It can be a reminiscence of events, and people who influenced those events. It is useful to correlate such events and people with various stages of development and apply this knowledge to a current situation.
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Characterization of an Avian Leukosis Virus Isolate

Avian Diseases, 1966
An earlier publication reported the screening of hens from a commercial flock (Spring Lake Farms) for the purpose of selecting dams with which to establish a flock free of infection with agents possessing RIF (Resistance Inducing Factor) activity, i.e. the induction of cell resistance to Rous sarcoma virus (RSV) (6).
S, Levine, D, Nelsen, W, Olson
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Restrictions That Influence Avian Leukosis Virus-Induced Lymphoid Leukosis

1986
Infection of one day old White leghorn chicks with avian leukosis virus (ALV) usually results in the development of a bursal- dependent B-cell lymphoma (Purchase and Burmester, 1978). This tumor requires the bursal environment to develop and is characterized by the presence of cell surface immunoglobulin M (IgM) (Cooper et al., 1974).
E H, Humphries, T W, Baba
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Genetics of Leukemogenesis by Avian Leukosis Viruses

1981
Oncoviruses of chickens can be classified into one of three groups, depending on their effects on cells in culture and their pathogenicity and lifestyle in the chicken (Fig. 1). The most intensely studied but rarest of these viruses are the transforming acute leukemia and sarcoma viruses.
J M, Coffin, P N, Tsichlis, H L, Robinson   +2 more
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On the Terminology and Classification of the Avian Leukosis Complex

Avian Diseases, 1967
Recent advances in studies of the causes and pathogenesis of a group of neoplasms heretofore known as the avian leukosis complex have provoked a reconsideration of established terms and classifications. Several classifications (1,2,5,6,7,10), each a product and a contribution of its time, have been published in the literature.
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Genes, Viruses, and Avian Leukosis

BioScience, 1981
Viral induction of leukemia and solid tumors in the chicken were reported in the first decade of the 20th century by Ellerman and Bang (1908, 1909) in Denmark and Rous (1911) in the United States. Over the next 50 years, a small group of workers studied a variety of virus-induced neoplasms in the chicken and developed a number of sarcoma and acute ...
L. B. Crittenden, S. M. Astrin
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Avian Lymphoid Leukosis: Mechanisms of Lymphomagenesis

1988
Publisher Summary This chapter discusses the factors responsible for the progression of the neoplastic condition through each stage in the ontogeny of lymphoid leukosis (LL), namely, infection, hyperplastic transformation, bursal lymphoma development, and metastasis to visceral organs.
D L, Ewert, G F, de Boer
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Ribosomal rna in avian leukosis virus particles

International Journal of Cancer, 1971
AbstractThe RNAs of avian viruses originating from the plasma of leukemic chickens (AMV) or from tissue culture (MAV‐B) consisted of five distinct components with sedimentation constants of 62 s, 27 s, 17 s, 9 s, and 4–5 s. Whereas the 27 s and 17 s minor RNA components could be consistently recovered from large quantities of AMV, fractionation ...
T, Obara, D P, Bolognesi, H, Bauer
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