Results 171 to 180 of about 7,272 (223)

Genes, Viruses, and Avian Leukosis

BioScience, 1981
Viral induction of leukemia and solid tumors in the chicken were reported in the first decade of the 20th century by Ellerman and Bang (1908, 1909) in Denmark and Rous (1911) in the United States. Over the next 50 years, a small group of workers studied a variety of virus-induced neoplasms in the chicken and developed a number of sarcoma and acute ...
L. B. Crittenden, S. M. Astrin
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Avian Lymphoid Leukosis: Mechanisms of Lymphomagenesis

1988
Publisher Summary This chapter discusses the factors responsible for the progression of the neoplastic condition through each stage in the ontogeny of lymphoid leukosis (LL), namely, infection, hyperplastic transformation, bursal lymphoma development, and metastasis to visceral organs.
D L, Ewert, G F, de Boer
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Characterization of an Avian Leukosis Virus Isolate

Avian Diseases, 1966
An earlier publication reported the screening of hens from a commercial flock (Spring Lake Farms) for the purpose of selecting dams with which to establish a flock free of infection with agents possessing RIF (Resistance Inducing Factor) activity, i.e. the induction of cell resistance to Rous sarcoma virus (RSV) (6).
S, Levine, D, Nelsen, W, Olson
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Ribosomal rna in avian leukosis virus particles

International Journal of Cancer, 1971
AbstractThe RNAs of avian viruses originating from the plasma of leukemic chickens (AMV) or from tissue culture (MAV‐B) consisted of five distinct components with sedimentation constants of 62 s, 27 s, 17 s, 9 s, and 4–5 s. Whereas the 27 s and 17 s minor RNA components could be consistently recovered from large quantities of AMV, fractionation ...
T, Obara, D P, Bolognesi, H, Bauer
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Pathogenicity of Two Recombinant Avian Leukosis Viruses

Avian Diseases, 2003
We have recently described the isolation and molecular characteristics of two recombinant avian leukosis subgroup J viruses (ALV J) with an avian leukosis virus subgroup A envelope (r5701A and r6803A). In the present study, we examined the role of the subgroup A envelope in the pathogenesis of these recombinant viruses.
Blanca, Lupiani, Susan M, Williams, Robert F, Silva, Henry D, Hunt, Aly M, Fadly   +4 more
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Genetics of Leukemogenesis by Avian Leukosis Viruses

1981
Oncoviruses of chickens can be classified into one of three groups, depending on their effects on cells in culture and their pathogenicity and lifestyle in the chicken (Fig. 1). The most intensely studied but rarest of these viruses are the transforming acute leukemia and sarcoma viruses.
J M, Coffin, P N, Tsichlis, H L, Robinson   +2 more
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Avian leukosis virus‐receptor interactions

Avian Pathology, 1998
Cellular receptors for subgroups A, B, D and E avian leukosis virus (ALV) have been identified and characterized. The Tva receptor for subgroup A ALV is a member of the low density lipoprotein receptor family of proteins. There is an accumulating body of evidence to suggest that this receptor binds specifically to subgroup A viral envelope (Env ...
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Yellow Fever Vaccine and Avian Leukosis Virus

Annals of Internal Medicine, 1984
Excerpt To the editor: The recommendations of the Immunization Practices Advisory Committee for yellow fever immunization have recently been updated again (1).
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Progression of Avian Lymphoid Leukosis

1989
The leukosis/sarcoma viruses, members of the avian oncornavirus or oncovirus group, cause a wide spectrum of tumors in chickens. The most common under field conditions is lymphoid leukosis, a B-cell lymphoma. In susceptible chickens infected at an early age, cells of the cortex of the bursa of Fabricius of 6- to 8-week-old chickens transform and ...
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Developments in avian leukosis research.

Leukemia, 1992
Infection by exogenous avian leukosis viruses (ALVs) causes economic loss from neoplastic mortality and from impaired performance of subclinically infected chickens. This paper reviews progress in research related to natural infection and its control.
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