Results 201 to 210 of about 6,163 (232)
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Studies on the avian leukosis complex
Journal of Comparative Pathology and Therapeutics, 1950Summary A fall in the tumour producing activity of suspensions of avian lymphoid tumours occurs after freezing to −70°C and subsequent thawing. Dextrose exerts a protective effect in that it considerably reduces the fall in activity. Suspensions tested, at two separate intervals after storage at −70°C have shown no significant differences in ...
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Progression of Avian Lymphoid Leukosis
1989The leukosis/sarcoma viruses, members of the avian oncornavirus or oncovirus group, cause a wide spectrum of tumors in chickens. The most common under field conditions is lymphoid leukosis, a B-cell lymphoma. In susceptible chickens infected at an early age, cells of the cortex of the bursa of Fabricius of 6- to 8-week-old chickens transform and ...
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Yellow Fever Vaccine and Avian Leukosis Virus
Annals of Internal Medicine, 1984Excerpt To the editor: The recommendations of the Immunization Practices Advisory Committee for yellow fever immunization have recently been updated again (1).
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Characterization of Monoclonal Antibodies to Avian Leukosis Viruses
Avian Diseases, 1986Hybridoma cell lines secreting monoclonal antibody (MCA) to avian leukosis virus (ALV) structural proteins p27 and p19 have been established. In an indirect enzyme-linked immunosorbent assay (ELISA), MCA 6AL20 (IgG1 isotype) reacted with RPL-40 (ALV subgroup A), avian myeloblastosis virus (AMV) (a mixture of subgroups A and B), Rous-associated virus ...
L. B. Crittenden +4 more
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Immunology of Avian Leukosis Virus Infections [PDF]
, 1986 This Chapter will concentrate on three interactions of avian leukosis virus (ALV) with the immune system of the avian host. The first, neoplastic transformation of lymphoid cells, will be treated briefly and is the subject of more detailed discussion in Chapters 4 and 9 of this monograph.
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The Incidence of Avian Leukosis in Broilers at Processing
Avian Diseases, 1962sex of the involved bird. Such observations were tabulated to obtain a breakdown of the various forms of leukosis. The condemnation data were obtained from the inspector's records to determine over-all condemnation rate and the categories involved.
W. J. Benton, W. C. Krauss, M. S. Cover
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Avian leukosis virus‐receptor interactions
Avian Pathology, 1998Cellular receptors for subgroups A, B, D and E avian leukosis virus (ALV) have been identified and characterized. The Tva receptor for subgroup A ALV is a member of the low density lipoprotein receptor family of proteins. There is an accumulating body of evidence to suggest that this receptor binds specifically to subgroup A viral envelope (Env ...
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Epizootiology of Avian Leukosis Virus Infections
1986This Chapter is concerned with the spread and survival of avian leukosis virus (ALV) in populations of domestic fowl, and the various factors which influence infection rate and incidence of disease. The term ALV as used here denotes the common “wild-type” exogenous avian retrovirus which causes mainly lymphoid leukosis (LL).
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Approaches to Control Avian Lymphoid Leukosis
1987In the past, the eradication of lymphoid leukosis (LL), the most common pathological manifestation of avian leukosis virus (ALV) infections in chickens, could only be pursued by elimination of all diseased birds from a given flock. This approach was only successful if the initial incidence of ALV infection was low.
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