Results 131 to 140 of about 5,611 (177)
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Plaque assay for some strains of avian leukosis virus

Virology, 1972
Abstract Some strains of avian leukosis virus were found to produce plaques at 41 ° in cultures of chick embryo cells which had been fully infected with a temperature-sensitive mutant of Rous sarcoma virus (RSV) which fails to induce morphological transformation at the elevated temperature.
S, Kawai, H, Hanafusa
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Hypergammaglobulinemia in Chickens Congenitally Infected with an Avian Leukosis Virus

The Journal of Immunology, 1976
Abstract Significantly elevated (2- to 5-fold higher than controls) serum levels of IgG were found in chickens congenitally infected with F42 strain of avian leukosis (ALV-F42), a subgroup A avian leukosis virus (ALV). A further increase in IgG levels in congentially infected birds was found to be induced by injection of influenza virus ...
L F, Qualtiere, P, Meyers
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5′ avian leukosis virus sequences and osteopetrotic potential

Virology, 1992
Recombinants of Rous-associated virus-0 and Br21 have been used to localize 5' viral sequences that affect the osteopetrotic potential of avian leukosis viruses. Rous-associated virus-0 is a benign subgroup E virus of endogenous origin that does not cause osteopetrosis. Br21 is a constructed subgroup E virus with high osteopetrotic potential.
Robinson, Harriet L.   +5 more
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Serologic Survey of Man for Avian Leukosis Virus Infection

The Journal of Immunology, 1967
Summary Six current lots of 17 D yellow fever virus vaccine propagated in chicken embryos were found to be contaminated with complement-fixing group antigens of avian leukosis virus. Bloods of 111 individuals vaccinated with 22 different lots of the 17 D vaccine given up to 54 months before drawing a blood specimen were ...
F, Piraino   +2 more
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Occurrence of subgroup J avian leukosis virus in Taiwan

Avian Pathology, 2002
There are three grandparent farms for three different chicken breeds in Taiwan. One of these farms, populated by breast meat yield chickens (yield type), suffered from a severe subgroup J avian leukosis virus (ALV-J) infection in mid-1997. The affected flocks at that farm had a weekly mortality of more than 1% and a 15% drop in egg production.
C-H, Wang, Y-W, Juan
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The problem of the phage-like structure of the avian leukosis virus

Experientia, 1963
Fur das Vorhandensein der spheroidalen und der phag-ahnlichen Form des Huhner-Leukosis-Virus (BAI, Stamm A) durfte das Stadium des Leukamieprozesses, metabolische Eigenschaften der Myeloblasten sowie deren Milieu verantwortlich sein.
P, BARTL, J, RIMAN, F, SORM
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Avian leukosis virus‐receptor interactions

Avian Pathology, 1998
Cellular receptors for subgroups A, B, D and E avian leukosis virus (ALV) have been identified and characterized. The Tva receptor for subgroup A ALV is a member of the low density lipoprotein receptor family of proteins. There is an accumulating body of evidence to suggest that this receptor binds specifically to subgroup A viral envelope (Env ...
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Detection of avian leukosis virus: comparison of five techniques

Research in Veterinary Science, 1984
Five techniques were compared for their ability to detect decreasing dilutions of RAV-I, an avian leukosis sarcoma virus, in serially passaged chick embryo fibroblast cell cultures. The indirect fluorescent antibody test, sandwich enzyme-linked immunosorbent assay (ELISA) and reverse transcriptase assay were equally sensitive in detecting the virus ...
R A, Nicholas, D H, Thornton
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Myotropic Avian Leukosis Virus subgroup J Infection in a Chicken

Journal of Veterinary Medicine, Series B, 2006
SummaryThe study describes a highly productive myotropic avian leukosis virus infection (ALV) in a 3‐month‐old female chicken. At necropsy, ascites, hepatic fibrosis and cardiomegaly were seen. Histologically, the most striking lesion was the presence of cytoplasmic basophilic inclusions in myocardial fibers.
G. Sironi   +4 more
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Epizootiology of Avian Leukosis Virus Infections

1986
This Chapter is concerned with the spread and survival of avian leukosis virus (ALV) in populations of domestic fowl, and the various factors which influence infection rate and incidence of disease. The term ALV as used here denotes the common “wild-type” exogenous avian retrovirus which causes mainly lymphoid leukosis (LL).
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