Results 181 to 190 of about 1,004,755 (217)
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Hypergammaglobulinemia in Chickens Congenitally Infected with an Avian Leukosis Virus
The Journal of Immunology, 1976Abstract Significantly elevated (2- to 5-fold higher than controls) serum levels of IgG were found in chickens congenitally infected with F42 strain of avian leukosis (ALV-F42), a subgroup A avian leukosis virus (ALV). A further increase in IgG levels in congentially infected birds was found to be induced by injection of influenza virus ...
L F, Qualtiere, P, Meyers
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Serologic Survey of Man for Avian Leukosis Virus Infection
The Journal of Immunology, 1967Summary Six current lots of 17 D yellow fever virus vaccine propagated in chicken embryos were found to be contaminated with complement-fixing group antigens of avian leukosis virus. Bloods of 111 individuals vaccinated with 22 different lots of the 17 D vaccine given up to 54 months before drawing a blood specimen were ...
F, Piraino +2 more
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The problem of the phage-like structure of the avian leukosis virus
Experientia, 1963Fur das Vorhandensein der spheroidalen und der phag-ahnlichen Form des Huhner-Leukosis-Virus (BAI, Stamm A) durfte das Stadium des Leukamieprozesses, metabolische Eigenschaften der Myeloblasten sowie deren Milieu verantwortlich sein.
P, BARTL, J, RIMAN, F, SORM
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Avian leukosis virus‐receptor interactions
Avian Pathology, 1998Cellular receptors for subgroups A, B, D and E avian leukosis virus (ALV) have been identified and characterized. The Tva receptor for subgroup A ALV is a member of the low density lipoprotein receptor family of proteins. There is an accumulating body of evidence to suggest that this receptor binds specifically to subgroup A viral envelope (Env ...
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Occurrence of subgroup J avian leukosis virus in Taiwan
Avian Pathology, 2002There are three grandparent farms for three different chicken breeds in Taiwan. One of these farms, populated by breast meat yield chickens (yield type), suffered from a severe subgroup J avian leukosis virus (ALV-J) infection in mid-1997. The affected flocks at that farm had a weekly mortality of more than 1% and a 15% drop in egg production.
C-H, Wang, Y-W, Juan
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Detection of avian leukosis virus: comparison of five techniques
Research in Veterinary Science, 1984Five techniques were compared for their ability to detect decreasing dilutions of RAV-I, an avian leukosis sarcoma virus, in serially passaged chick embryo fibroblast cell cultures. The indirect fluorescent antibody test, sandwich enzyme-linked immunosorbent assay (ELISA) and reverse transcriptase assay were equally sensitive in detecting the virus ...
R A, Nicholas, D H, Thornton
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Myotropic Avian Leukosis Virus subgroup J Infection in a Chicken
Journal of Veterinary Medicine, Series B, 2006SummaryThe study describes a highly productive myotropic avian leukosis virus infection (ALV) in a 3‐month‐old female chicken. At necropsy, ascites, hepatic fibrosis and cardiomegaly were seen. Histologically, the most striking lesion was the presence of cytoplasmic basophilic inclusions in myocardial fibers.
G. Sironi +4 more
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Epizootiology of Avian Leukosis Virus Infections
1986This Chapter is concerned with the spread and survival of avian leukosis virus (ALV) in populations of domestic fowl, and the various factors which influence infection rate and incidence of disease. The term ALV as used here denotes the common “wild-type” exogenous avian retrovirus which causes mainly lymphoid leukosis (LL).
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Immunology of Avian Leukosis Virus Infections
1986This Chapter will concentrate on three interactions of avian leukosis virus (ALV) with the immune system of the avian host. The first, neoplastic transformation of lymphoid cells, will be treated briefly and is the subject of more detailed discussion in Chapters 4 and 9 of this monograph.
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Contact transmission of avian leukosis virus.
Journal of the National Cancer Institute, 1977Intravenous inoculation of four age groups of White Leghorn chicks with ALV-F42, a group A field strain of avian leukosis virus (ALV), indicated that persistent tolerant infection could be induced as late as 2 weeks post hatch, though most birds responded with neutralizing antibody.
K G, Weyl, R M, Dougherty
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