Results 121 to 130 of about 3,390 (167)
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Bartter's syndrome and erythrocytosis
The American Journal of Medicine, 1973Abstract We describe a patient, known since 1960, with Bartter's syndrome and erythrocytosis. The diagnosis was made on the basis of hypokalemic alkalosis, hyperplasia of the juxtaglomerular apparatus, resistance to the presser effect of angiotensin, extremely high plasma renin activity and normal blood pressure.
D W, Erkelens, L W, Statius van Eps
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Current Opinion in Nephrology and Hypertension, 2003
This review describes recent advances in our understanding of the genetic heterogeneity, pathophysiology and treatment of Bartter syndrome, a group of autosomal recessive disorders that are characterized by markedly reduced or absent salt transport by the thick ascending limb of Henle.
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This review describes recent advances in our understanding of the genetic heterogeneity, pathophysiology and treatment of Bartter syndrome, a group of autosomal recessive disorders that are characterized by markedly reduced or absent salt transport by the thick ascending limb of Henle.
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Bartter and Gitelman Syndromes
Advances in Pediatrics, 2000Since the initial description in the 1960s of patients with seemingly inherited disorders characterized by hypokalemia and metabolic alkalosis, the pathophysiologic processes underlying Bartter and Gitelman syndromes have generated tremendous study and speculation.
S J, Schurman, L R, Shoemaker
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Pathogenesis of Bartter's Syndrome
New England Journal of Medicine, 1969Abstract In a patient with Bartter's syndrome (increased plasma renin, juxtaglomerular-cell hyperplasia, hyperaldosteronism and hypokalemia, but no hypertension), aldosterone excretion and secretion were increased only moderately despite marked elevation of plasma renin, presumably because of suppression of aldosterone production by hypokalemia.
A D, Goodman +2 more
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Bartter's syndrome and pregnancy
European Journal of Obstetrics & Gynecology and Reproductive Biology, 1999Bartter's syndrome is a rare renal tubular disorder and few cases are reported associated with pregnancy. We describe a case of long standing disease with a good obstetric outcome in which the main concern was to maintain normal potassium serum levels.
N, Clode +6 more
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Nephrocalcinosis in Bartter's syndrome
Pediatric Nephrology, 1988Nephrocalcinosis was demonstrated by computerized tomography (CT) in all five children with Bartter's syndrome followed at our institution. In three of these five patients, nephrocalcinosis was also noted on ultrasound examination. Hypercalciuria was present in only one case. The mechanism leading to renal calcification remains unclear in this disease.
L, Garel, D, Filiatrault, P, Robitaille
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Archives of Internal Medicine, 1982
Two sisters were found to have Bartter's syndrome. Both had hypokalemia, hyperreninemia, normal BPs, and decreased pressor responses to angiotensin II. During a water diuresis, patient 1 had an abnormally low distal tubular fractional reabsorption of chloride initially, but this normalized after hypokalemia was corrected for one year.
T, Ogihara +5 more
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Two sisters were found to have Bartter's syndrome. Both had hypokalemia, hyperreninemia, normal BPs, and decreased pressor responses to angiotensin II. During a water diuresis, patient 1 had an abnormally low distal tubular fractional reabsorption of chloride initially, but this normalized after hypokalemia was corrected for one year.
T, Ogihara +5 more
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Inheritance of Bartter syndrome
American Journal of Medical Genetics, 1983AbstractThe Bartter syndrome is regarded as an autosomal recessive trait because of sib occurrence, equal sex ratio, and normal parents. Recently, obligatory carriers were shown to have the same pattern of platelet aggregation inhibition as their affected children, possibly a reflection of altered prostaglandin action.
R. Rodrigues Pereira +2 more
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Urinary Prostaglandins in Bartter's and Pseudo-Bartter's Syndrome
Uremia Investigation, 1985Renal secretion of prostaglandins (PG) in Bartter's syndrome and in different forms of hypokalemic normotensive syndromes (pseudo-Bartter's syndrome) was measured to determine if it is possible to use the measurement of urinary prostaglandins for the discrimination of different etiologies.
A, Hornych, C, Krief, J, Aumont
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Simulacra of Bartter's Syndrome
JAMA: The Journal of the American Medical Association, 1980A recent issue of Nephron (23:57-58, 1979) pays tribute to Dr Frederick Crosby Bartter on the occasion of his retirement, after 27 years of service as Chief of Endocrinology Section of the National Heart Institute, to become Chief of Staff for Research at the Veterans Administration Hospital in San Antonio, Tex.
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