Results 311 to 320 of about 484,763 (358)
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Erythrocytes and Blood Coagulation
Thrombosis and Haemostasis, 1974SummaryThe action of intact and hemolyzed red cells in in vitro clotting systems is analyzed against the background of the clinical manifestations of red cell procoagulants in incompatible transfusions and hemolytic anemias in man and in experimental animals.
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The Coagulation of Blood and Hemostasis
Annual Review of Physiology, 1950Since the number of papers on coagulation has greatly in creased, the reviewer, in order to avoid merely cataloging them with telegraphic brevity, was compelled to select mainly those which contribute specifically to the physiology of blood clotting.
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Regulation of blood coagulation
Biochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology, 2000The protein C anticoagulant pathway converts the coagulation signal generated by thrombin into an anticoagulant response through the activation of protein C by the thrombin-thrombomodulin (TM) complex. The activated protein C (APC) thus formed interacts with protein S to inactivate two critical coagulation cofactors, factors Va and VIIIa, thereby ...
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Transfusion and Apheresis Science, 2018
Hemostasis is the physiological control of bleeding and is initiated by subendothelial exposure. Platelets form the primary vascular seal in three stages (localization, stimulation and aggregation), which are triggered by specific interactions between platelet surface receptors and constituents of the subendothelial matrix.
Edward L G, Pryzdial +5 more
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Hemostasis is the physiological control of bleeding and is initiated by subendothelial exposure. Platelets form the primary vascular seal in three stages (localization, stimulation and aggregation), which are triggered by specific interactions between platelet surface receptors and constituents of the subendothelial matrix.
Edward L G, Pryzdial +5 more
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The Genetics of Blood Coagulation
1983Hemostasis is the term applied to the process that regulates the loss of blood from the circulatory system following injury. It involves three interrelated physiological mechanisms: constriction of blood vessels, aggregation of blood platelets to damaged subendothelial surfaces, and the formation of fibrin clots.
J B, Graham +3 more
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Prostacyclin and Blood Coagulation
Drugs, 1981Prostacyclin is a potent but unstable vasodilator, and inhibitor of platelet aggregation, which is produced by blood vessel walls. Platelet aggregability may be constantly conditioned in vivo by local or circulating prostacyclin. Prostacyclin is important in the maintenance of vascular homeostasis and may be implicated in certain disease states.
S, Moncada, J R, Vane
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Clinics in Laboratory Medicine, 1996
This article reviews the current understanding of coagulation and its role in overall hemostasis. Also provided is an overview of current screening and specialized tests of coagulation.
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This article reviews the current understanding of coagulation and its role in overall hemostasis. Also provided is an overview of current screening and specialized tests of coagulation.
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Supercharged blood coagulation
Blood, 2009In this issue of Blood, Undas and colleagues show that patients with AMI are able to mount an enhanced blood clotting response in the peripheral microvasculature.
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Hemodialysis International, 2001
The endothelium is the principal anti‐thrombotic mechanism, providing a non wettable surface and generating potent vasodilators (nitric oxide and prostacyclin) and clotting inhibitors [thrombomodulin and tissue factor pathway inhibitor (TFPI)]. When the integrity of the endothelium is breached, vasoconstriction occurs through neural and chemical ...
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The endothelium is the principal anti‐thrombotic mechanism, providing a non wettable surface and generating potent vasodilators (nitric oxide and prostacyclin) and clotting inhibitors [thrombomodulin and tissue factor pathway inhibitor (TFPI)]. When the integrity of the endothelium is breached, vasoconstriction occurs through neural and chemical ...
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Journal of Biomedical Materials Research, 1968
AbstractThe effect of phenol and certain phenyl compounds on the different stages of the blood coagulation and fibrinolytic mechanism is investigated. Phenol has a complex action on blood coagulation. In an optimal concentration of 0.3% it produced a definite, though suboptimal platelet factor 3 activity, evolved factor XII, accelerated thrombin ...
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AbstractThe effect of phenol and certain phenyl compounds on the different stages of the blood coagulation and fibrinolytic mechanism is investigated. Phenol has a complex action on blood coagulation. In an optimal concentration of 0.3% it produced a definite, though suboptimal platelet factor 3 activity, evolved factor XII, accelerated thrombin ...
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