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Antigenic variation in Borrelia
Research in Microbiology, 1991Antigenic variation was demonstrated for the agent of relapsing fever, Borrelia hermsii. The phenomenon is correlated with changes in major surface proteins called Vmp. The genes encoding these antigens are located on linear plasmids. Expression occurs by transposition of genes encoding Vmp to a telomeric expression site located on another linear ...
I, Saint Girons, A G, Barbour
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Borrelia Infection in Children
Acta Paediatrica, 1989ABSTRACT. All children (15 years) admitted during 1986 to Sachs Children's Hospital and presenting signs of facial palsy and/or meningitis, or with a history of known tick bite followed by headache, fatigue and muscle pain, were investigated for antibodies to Borrelia in serum and cerebrospinal fluid.
A S, Naglo, K, Wide
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Borrelia infection and vertigo
Acta Oto-Laryngologica, 198873 patients with vertigo were studied regarding serum antibodies to Borrelia spirochete antigen, using an indirect immunofluorescence method. Ten patients (14%) had serological evidence of Borrelia infection. All 10 patients had severe, incapacitating vertigo. Four of the Borrelia patients had positional vertigo and all 10 had positional nystagmus when
U, Rosenhall, P, Hanner, B, Kaijser
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Zentralblatt für Bakteriologie, Mikrobiologie und Hygiene. Series A: Medical Microbiology, Infectious Diseases, Virology, Parasitology, 1986
Antibodies to Borrelia spirochetes were determined by an enzyme-linked immunosorbent assay (ELISA) in sera from 298 patients with postinfectious arthritis. Sera from healthy individuals, patients with acute infectious meningitis of proven etiology and patients with multiple sclerosis served as controls. Titers above the 100% percentile of controls were
G, Stiernstedt, M, Granström
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Antibodies to Borrelia spirochetes were determined by an enzyme-linked immunosorbent assay (ELISA) in sera from 298 patients with postinfectious arthritis. Sera from healthy individuals, patients with acute infectious meningitis of proven etiology and patients with multiple sclerosis served as controls. Titers above the 100% percentile of controls were
G, Stiernstedt, M, Granström
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The Molecular Biology of Borrelia
Clinical Infectious Diseases, 1989Borrelia burgdorferi, the cause of Lyme disease, has two major outer-membrane proteins, OspA and OspB, which act as surface antigens. A 49-kilobase linear plasmid contains the genes that encode for these surface proteins. Direct examination of denatured plasmid molecules has revealed single-stranded circles with a circumference of approximately 100 ...
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Borrelia meningitis in Denmark
Zentralblatt für Bakteriologie, Mikrobiologie und Hygiene. Series A: Medical Microbiology, Infectious Diseases, Virology, Parasitology, 1987We studied 23 patients with borrelia meningitis, a tick-borne spirochetosis increasingly recognized in Denmark. The disease showed a seasonal incidence from June to December. Twenty one patients experienced a painful sensory radiculitis, in 17 cases followed by a predominantly cranial mononeuritis multiplex mainly affecting the facial nerve (Bannwarth ...
K, Hansen +4 more
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Zentralblatt für Bakteriologie, Mikrobiologie und Hygiene. Series A: Medical Microbiology, Infectious Diseases, Virology, Parasitology, 1986
DNA homology studies were performed on Borrelia hermsii, B. duttoni, B. crocidurae and the O. coriaceus spirochete. These organisms had a genus level relationship with each other. B. hermsii was 17 to 53% homologous with B. duttoni, B. crocidurae, and the O. coriaceus spirochetes; B. crocidurae was 17-32% related to B. hermsii, B.
F W, Hyde, R C, Johnson
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DNA homology studies were performed on Borrelia hermsii, B. duttoni, B. crocidurae and the O. coriaceus spirochete. These organisms had a genus level relationship with each other. B. hermsii was 17 to 53% homologous with B. duttoni, B. crocidurae, and the O. coriaceus spirochetes; B. crocidurae was 17-32% related to B. hermsii, B.
F W, Hyde, R C, Johnson
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Microbiology of Borrelia burgdorferi
Seminars in Neurology, 1997This article reviews the natural history, taxonomy, physical structure, growth requirements, and molecular structure of Borrelia burgdorferi sensu lato, the causative agent of Lyme disease. These spirochetal bacteria are maintained in nature through an infectious cycle between wild mammals and ticks.
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