Results 131 to 140 of about 19,348 (185)

The Flagellin-Specific Chaperone FliS of Borrelia burgdorferi Controls the Cytoplasmic Pool of Flagellins at the Level of Translation Initiation, Secretion, and Proteolysis. [PDF]

Mol Microbiol
Sze CW, Zhang K, Lynch MJ, Guo W, Liu J, Crane BR, Li C.   +6 more
europepmc   +1 more source

Borrelia burgdorferi lacking all cp32 prophage plasmids retains full infectivity in mice. [PDF]

EMBO Rep
Hillman C, Theriault H, Dmitriev A, Hansra S, Rosa PA, Wachter J.   +5 more
europepmc   +1 more source

Secretory leukocyte protease inhibitor influences periarticular joint inflammation in <i>Borrelia burgdorferi</i>-infected mice. [PDF]

Elife
Yu Q, Tang X, Hart T, Homer R, Belperron AA, Bockenstedt LK, Ring A, Nakamura A, Fikrig E.   +8 more
europepmc   +1 more source

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Asymptomatic Borrelia burgdorferi infection

Medical Hypotheses, 2001
Little is known about the natural history of asymptomatic Borrelia burgdorferi infection. Our analysis of the asymptomatic infections diagnosed serologically in a recent OspA vaccine trial conducted in the United States (N Engl J Med 1998;339: 209-215), suggests that the natural history of this event is more benign than that reported for untreated ...
G P, Wormser, R B, Nadelman, J, Nowakowski, I, Schwartz   +3 more
openaire   +2 more sources

Borrelia burgdorferi–Traveling incognito?

Microbes and Infection, 2006
We outline in this review how Borrelia burgdorferi, the causative agent of Lyme disease, moves from the tick to the vertebrate host, and what molecules are potentially involved in this challenging commute. The survival strategies utilized by the spirochete during transmission and the initial stages of infection are discussed.
Erol, Fikrig, Sukanya, Narasimhan
openaire   +2 more sources

Transformation of Borrelia burgdorferi

Current Protocols, 2021
AbstractTransformation techniques used to genetically manipulate Borrelia burgdorferi, the agent of Lyme disease, play a critical role in generating mutants that facilitate analyses of the role of genes in the pathophysiology of this bacterium.
J, Seshu, Brian E, Moy, Taylor MacMackin, Ingle   +2 more
openaire   +2 more sources

Borrelia burgdorferi and localized scleroderma

Clinics in Dermatology, 1994
L yme borreliosis is a multisystemic disorder caused by the spirochete Borrelia burgdorferi and transmitted by the Ixodes tick. The initial manifestations of the disease are the characteristic skin lesion, erythema migrans, and symptoms of general malaise (stage I).
TREVISAN, GIUSTO, D. H. E. REES, G. STINCO   +2 more
openaire   +4 more sources

Borrelia burgdorferi Microarray Analysis

2017
Microarray studies have contributed significantly to the current understanding of Borrelia burgdorferi genome content and transcriptional regulation. Here, we describe the use of microarray technology for several aspects of B. burgdorferi genomic analysis.
Radha, Iyer, Ira, Schwartz
openaire   +2 more sources

Nonprotein Antigens of Borrelia burgdorferi

Journal of Infectious Diseases, 1993
Preparative thin-layer chromatograms of chloroform-methanol extracts of Borrelia burgdorferi (B31) sonicates showed four fractions (Rf values of 0.84, 0.81, 0.66 and 0.61) that stained with iodine vapors, orcinol, or phospray, suggesting the presence of lipid-, carbohydrate-, and phosphorus-containing compounds.
C M, Wheeler, J C, Garcia Monco, J L, Benach, M G, Golightly, G S, Habicht, A C, Steere   +5 more
openaire   +2 more sources

Microbiology of Borrelia burgdorferi

Seminars in Neurology, 1997
This article reviews the natural history, taxonomy, physical structure, growth requirements, and molecular structure of Borrelia burgdorferi sensu lato, the causative agent of Lyme disease. These spirochetal bacteria are maintained in nature through an infectious cycle between wild mammals and ticks.
openaire   +2 more sources