Results 201 to 210 of about 534,808 (344)
[Brown tumor due to hyperparathyroidism secondary to chronic kidney disease: A clinical case]. [PDF]
Contreras-Vargas LM +9 more
europepmc +1 more source
Automated procedural analysis is recognized as one of the major game changers for robotic surgery. Meaning digital analysis needs to replace the manual assessments that set todays standard. Mechanical robotic‐instrument tracking enables the derivation of quantitative kinematic metrics that support behavior‐based workflow segmentation into distinct ...
Kateryna Pirkovets +4 more
wiley +1 more source
Primary Hyperparathyroidism-Induced Brown Tumor of the Mandible: A Diagnostic Dilemma. [PDF]
Boral D +4 more
europepmc +1 more source
A patient with multiple brown tumors due to secondary hyperparathyroidism: A case report
Laura Messina +6 more
openalex +1 more source
Predictive models successfully screen nanoparticles for toxicity and cellular uptake. Yet, complex biological dynamics and sparse, nonstandardized data limit their accuracy. The field urgently needs integrated artificial intelligence/machine learning, systems biology, and open‐access data protocols to bridge the gap between materials science and safe ...
Mariya L. Ivanova +4 more
wiley +1 more source
Functional cystic parathyroid adenoma with bilateral mandibular brown tumor: a case report. [PDF]
Al-Smair A +4 more
europepmc +1 more source
ABSTRACT Background Eosinophilic chronic rhinosinusitis with nasal polyps (eCRSwNP) is characterized by persistent sinonasal inflammation and marked eosinophilic infiltration. Although the relationship between eosinophils and NP formation has been extensively studied, the mechanisms governing eosinophil transepithelial migration into the nasal mucosa ...
Yeong‐In Jo +7 more
wiley +1 more source
Primary hyperparathyroidism presenting as a brown tumor in the mandible: A case report. [PDF]
Staouni IB +6 more
europepmc +1 more source
The interaction between mitofusin 2 (MFN2) and phosphofurin acidic cluster sorting protein 2 regulates the formation of mitochondria‐associated endoplasmic reticulum membranes (MAM). Overexpression of MFN2 can promote the formation of MAMs and inhibit inflammation and oxidative stress response in mammary epithelial cells.
Xiechen Zhou +7 more
wiley +1 more source

