Results 181 to 190 of about 21,846 (223)
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Erythrodermic bullous pemphigoid
Journal of the American Academy of Dermatology, 1993REFERENCES L Korman N, Pemphigus. J AM ACAD DERMATOL 1988; 18:1219-38. 2. Barthelemy H, Frappaz A, Cambazard F, et al. Treatment of nine cases of pemphigus vulgaris with cyclosporine. J AM ACAD DERMATOL 1988;18:1262-6, 3. ~evuz J. Plasmapheresis. In: Orfanos CE, Stadler R, GollDIck H, eds. Dermatology in five continents.
A, Saitoh +5 more
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Bullous pemphigoid in adolescence
Pediatric Dermatology, 2018AbstractBullous pemphigoid (BP) is the most common autoimmune blistering disease affecting the elderly but is quite rare in childhood. The majority of pediatric cases have been reported during early childhood. Adolescence is divided into three phases: early (10‐13 years), middle (14‐17), and late (18‐21).
Aikaterini Patsatsi +2 more
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Bullous pemphigoid and penicillamine
Clinical and Experimental Dermatology, 1996We describe a woman who developed bullous pemphigoid in association with penicillamine therapy. While pemphigus is a well-recognized complication of penicillamine, pemphigoid should also be considered in the differential diagnosis of bullous eruptions induced by this drug.
R, Weller, M I, White
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Eosinophils in bullous pemphigoid
Panminerva Medica, 2021Bullous pemphigoid (BP) is an autoimmune blistering disorder with substantial morbidity and mortality. BP is regarded as a disorder driven by IgG due to BP180 and BP230 IgG autoantibodies, yet, new advances highlight the function of eosinophils and IgE autoantibodies in BP.
Virginia A, Jones +2 more
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The immunopathology of bullous pemphigoid
Clinics in Dermatology, 1987Abstract The immunopathology of bullous pemphigoid features basement membrane zone (BMZ) deposition of immunoglobulin and complement and the presence of circulating immunoreactants. 1,2 The demonstration of linear subepidermal deposition of C3 and/or IgG is usually necessary to establish the diagnosis.
M J, Imber, G F, Murphy, R E, Jordon
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Bullous and cicatricial pemphigoid
Journal of Autoimmunity, 1991Bullous pemphigoid (BP) and cicatricial pemphigoid are blistering mucocutaneous diseases characterized by detachment of the overlying epithelium from its stroma. IgG and complement components are deposited in all affected tissue at the level of blister formation--through the lamina lucida of the epithelium. The primary antibody response is of the IgG 4
G J, Anhalt, L H, Morrison
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Pathogenesis of Bullous Pemphigoid
Dermatologic Clinics, 2011Bullous pemphigoid, the most common autoimmune blistering disease, is induced by autoantibodies against type XVII collagen. Passive transfer of IgG or IgE antibodies against type XVII collagen into animals has revealed not only the pathogenicity of these antibodies but also the subsequent immune responses, including complement activation, mast cell ...
Hideyuki, Ujiie +2 more
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Erythrodermic bullous pemphigoid
International Journal of Dermatology, 2001Case 1 An 87‐year‐old man, with a 2‐year history of a generalized exfoliative erythroderma involving approximately 90% of the body, presented with an onset of tense blisters symmetrically distributed on most of his body.Clinical examination revealed generalized erythema, edema, moderate lichenification, and scaling, with several tense and grouped ...
L. Amato +5 more
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The Pathophysiology of Bullous Pemphigoid
Clinical Reviews in Allergy & Immunology, 2007Bullous pemphigoid (BP) is a blistering skin disease characterized by an autoimmune response to 2 hemidesmosomal proteins within the dermal-epidermal junction, designated BP180 and BP230. While BP230 localizes intracellularly and associates with the hemidesmosomal plaque, BP180 is a transmembrane glycoprotein with an extracellular domain.
Michael, Kasperkiewicz +1 more
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[Physiopathology of bullous pemphigoid].
Annales de dermatologie et de venereologie, 2009International ...
Doffoel-Hantz, V. +5 more
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