Results 251 to 260 of about 4,737,690 (404)

Nephronectin (NPNT) is a Crucial Determinant of Idiopathic Pulmonary Fibrosis: Modulating Cellular Senescence via the ITGA3/YAP1 Signaling Axis

open access: yesAdvanced Science, EarlyView.
Through a comprehensive multi‐omics analysis, this study identifies a marked reduction in Nephronectin (NPNT) expression within fibrotic lung tissue. This reduction impairs the binding capability to the ITGA3 receptor, consequently causing YAP1 to persist in the cytoplasm, where it undergoes degradation.
Jiayu Guo   +20 more
wiley   +1 more source

Severe trauma associated cardiac failure. [PDF]

open access: yesScand J Trauma Resusc Emerg Med
Dietrich M   +4 more
europepmc   +1 more source

Acute Lymphoblastic Leukemia Presenting with Acute Decompensated Cardiac Failure.

open access: yesHeart Views, 2023
Abdelghani MS   +4 more
europepmc   +1 more source

High-output Cardiac Failure: A Forgotten Phenotype in Clinical Practice. [PDF]

open access: yesCurr Cardiol Rev, 2022
de Ávila DX   +3 more
europepmc   +1 more source

The GRK2/AP‐1 Signaling Axis Mediates Vascular Endothelial Dysfunction and Atherosclerosis Induced by Oscillatory Low Shear Stress

open access: yesAdvanced Science, EarlyView.
This study highlights GRK2 is a central mediator in OSS‐induced endothelial dysfunction. OSS activates GPCRs in endothelial cells, leading to GRK2 phosphorylation and the activation of AP‐1. AP‐1 induces inflammation, while also promoting NR4A1 expression and anchoring LKB1 in the nucleus, which suppresses AMPK activity. This cascade causes endothelial
Li‐Da Wu   +18 more
wiley   +1 more source

Suicidal behaviour among patients with congestive cardiac failure in a Nigerian teaching hospital. [PDF]

open access: yesS Afr J Psychiatr
Shofu-Akanji TO   +5 more
europepmc   +1 more source

Management of high-output cardiac failure caused by head and neck AVMs: Percutaneous suture-assisted ethanol and coil embolotherapy. [PDF]

open access: yesFront Cardiovasc Med, 2022
Wen MZ   +8 more
europepmc   +1 more source

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