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Dialysis-associated muscle cramps related to high-flow arteriovenous fistula and high cardiac output failure reversed with fistula ligation. [PDF]

Clin Kidney J
Wahba IM.
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Hyperpyrexia Complicating Low Fixed Cardiac Output

Chest, 1977
A 41-year-old woman with end-stage mitral and aortic stenosis developed the cycle of low cardiac output, peripheral vasoconstriction, increased core temperature, and cardiac decompensation. This was interrupted by core cooling utilizing iced gastric lavage. We propose that her severe hyperthermia was caused or abetted by her low cardiac output.
T J, Salm, J P, Howe, J E, Dalen
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Treatment of perioperative low cardiac output syndrome

The Annals of Thoracic Surgery, 1995
New approaches to the treatment of perioperative low cardiac output are considered. In particular, use of the phosphodiesterase III inhibitors and their cardiovascular actions are reviewed and contrasted with those of conventional inotropic agents.
A R, Doyle, A K, Dhir, A H, Moors, R D, Latimer   +3 more
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Low Cardiac Output Syndrome: Identification and Management

Critical Care Nursing Clinics of North America, 2005
Low cardiac output syndrome (LCOS) is a clinical condition that is caused by a transient decrease in systemic perfusion secondary to myocardial dysfunction. The outcome is an imbalance between oxygen delivery and oxygen consumption at the cellular level which leads to metabolic acidosis. Although LCOS is observed most commonly in patients after cardiac
Linda, Massé, Marie, Antonacci
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Low Cardiac Output Following Cardiac Surgery

Dimensions of Critical Care Nursing, 1997
Patients often experience low cardiac output following cardiac surgery and as many as 90% of patients experience a decreased left ventricular ejection fraction (LVEF) and cardiac index (CI). Causes may vary from volume depletion to global myocardial dysfunction.
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Peripheral Vasodilators in Low Cardiac Output States

Surgical Clinics of North America, 1975
Vasodilators such as sodium nitroprusside and phentolamine have been used to reduce the afterload of the damaged and failing heart and increase the cardiac output. Only by understanding the basic physiology and how it may be altered can these agents be used appropriately in the care of the critically ill.
J S, Forrester, L, Protasio, K, Chatterjee   +2 more
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