Results 101 to 110 of about 518,643 (317)
Management of the Cardiac Patient Requiring Major Surgery [PDF]
Stanford Wessler, Herrman L. Blumgart
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Magnetoelectric nanoparticles exploit cancer cells' electrical vulnerabilities—altered membrane charge and higher capacitance—to achieve tumor‐specific targeting without bioreagents. MATLAB simulations reveal how electromagnetic forces create targeting specificity, while external magnetic fields enhance precision. This may enable wirelessly controlled,
Max Shotbolt+3 more
wiley +1 more source
ASSISTED CIRCULATION FOR CARDIAC FAILURE FOLLOWING INTRACARDIAC SURGERY WITH CARDIOPULMONARY BYPASS
Frank C. Spencer+3 more
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In this study, Acod1 knockout in CLP mice significantly increases peripheral blood NET levels, exacerbating inflammation, organ damage, and reducing survival. Further research shows that UBR5 interacts with PAD4, a key NET formation protein. Acod1/itaconate (ITA) enhances the enzymatic activity of UBR5 by alkylating the Cys2768 site, promoting the K48 ...
Huifan Liu+10 more
wiley +1 more source
Evaluation of Results of Cardiac Surgery-Supplement to the July Issue
Lewis Dexter
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Myocardial ischaemia and defects of atrioventricular conduction associated with cardiac surgery. [PDF]
David E. Taylor, C. F. Hider
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These findings suggest that UCP2 promotes LD formation and release in acinar cells by upregulating ACSL3 expression. This alteration in the local lipid metabolic environment indirectly drives the MMT process. Additionally, UCP2 may regulate the acetylation of Smad3 through Sirt1, enhancing its nuclear activity and activating the TGF‐β/ Smad3 signaling ...
Kunpeng Wang+9 more
wiley +1 more source
Sutureless and Rapid Deployment Prosthesis in Redo-Bentall Endocarditis
Aortic valve replacement (AVR) in a patient with a bio-Bentall conduit can be very challenging, especially if there was a previous endocarditis process for significant morbidity and mortality.
Salvatore Nicolardi+4 more
doaj +1 more source
What Social Services Offer to Patients Who Undergo Cardiac Surgery [PDF]
Roberta E. Peay
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CCDC183‐AS1 overexpression enhanced the ability of PCa cells to spread to the bone by inducing osteoclastogenesis. Mechanistically, CCDC183‐AS1 interacted with FUBP1 and enhanced its stability, which promoted the transcription of TNFSF14 (LIGHT). Copy number gain‐induced KDM5C epigenetically upregulated CCDC183‐AS1 expression by recruiting TET1 to the ...
Chuandong Lang+10 more
wiley +1 more source