Results 221 to 230 of about 162,067 (319)

Identification of Protein Networks and Biological Pathways Driving the Progression of Atherosclerosis in Human Carotid Arteries Through Mass Spectrometry-Based Proteomics. [PDF]

open access: yesInt J Mol Sci
Kalló G   +11 more
europepmc   +1 more source

Defining the Timing of Radiological Follow‐Up for Detection of Cholesteatoma Recidivism: A Meta‐analysis

open access: yesOtolaryngology–Head and Neck Surgery, EarlyView.
Abstract Objective Non‐echo‐planar diffusion‐weighted imaging (non‐EPI DWI) magnetic resonance imaging (MRI) has revolutionized postoperative cholesteatoma follow‐up, shifting from surgical to radiological evaluation. Yet, the timing for postoperative imaging remains contentious.
Amit Wolfovitz   +4 more
wiley   +1 more source

A review of histopathologic and radiologic features of non-atherosclerotic pathologies of the extracranial carotid arteries. [PDF]

open access: yesNeuroradiol J
Larson AS   +7 more
europepmc   +1 more source

Minimally invasive sacroiliac joint fusion: Primary endpoint results from the prospective, multicenter STACI study

open access: yesPM&R, EarlyView.
Abstract Background Chronic sacroiliac joint (SIJ) pain is highly debilitating. Minimally invasive SIJ fusion has become a commonly performed surgical treatment for SIJ pain, with level I evidence showing clinically significant improvements in pain, function, and quality of life and a low adverse event rate.
Jacqueline Weisbein   +11 more
wiley   +1 more source

Early and Long-Term Results of Simultaneous and Staged Revascularization of Coronary and Carotid Arteries. [PDF]

open access: yesPathophysiology
Golukhova E   +10 more
europepmc   +1 more source

Amentoflavone, a Preferentially TGF‐β Receptor 1 Inhibitor, Alleviates Cardiac Remodeling and Dysfunction Through Modulating the TGF‐β/Smad and MAPK Signaling Pathways

open access: yesPhytotherapy Research, EarlyView.
Amentoflavone, a preferentially TGF‐ββ receptor 1 inhibitor, attenuates transverse aortic constriction (TAC)‐triggered cardiac hypertrophy, fibrosis, and dysfunction by suppressing downstream Smad and MAPK signaling cascades in cardiomyocytes and cardiac fibroblasts.
Jiangjiao Wu   +10 more
wiley   +1 more source

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