Results 31 to 40 of about 27,915 (284)

Lymphoadenopathies caused by Bartonella henselae

open access: yesMedičnì Perspektivi, 2023
Bartonella henselae is the causative agent of cat scratch disease, the main manifestation of which is lymphadenopathy, but it can manifest as bacillary angiomatosis, injury to internal organs, joints, eyes, heart, kidneys.
V.S. Zhukovskiy   +3 more
doaj   +1 more source

HUMAN BARTONELLA INFECTION: A REVIEW OF LITERATURE [PDF]

open access: yesJournal of IMAB, 2021
Cat scratch disease has been reported in the literature for more than half a century as a syndrome of regional lymphadenopathy and fever. However, only a quarter of a century has passed since Bartonella henselae was identified as an etiological agent. As
Bistra Blagova, Nikolay Yanev
doaj   +1 more source

EPIDEMIOLOGICAL STUDIES ON OCULAR CAT-SCRATCH-DISEASE [PDF]

open access: yesVeterinary Medical Journal - Giza, 1995
Outpatients visiting ophthalmic clinics of Kasr  El-Eini hospitals and Research Institute of Ophthalmology (n=138) were examined for the presence of CSD. The clinical diagnosis of CSD was made in 14.49% of the examined patient on the bases of Hanger-Rose
WAFAA REDA, OMEIMA MOSTAFA
doaj   +1 more source

Neurologic Complications of Cat-Scratch Disease

open access: yesPediatric Neurology Briefs, 1991
The neurologic complications of cat-scratch disease in 76 patients with an average age of 10 years are reported from the Jackson-ville Health Education Program, University of Florida, and Department of Pediatrics, Uniformed Services, Bethesda, MD.
J Gordon Millichap
doaj   +1 more source

An unusual case of cat scratch disease

open access: yesClinical Management Issues, 2015
A 53-year-old man arrives in emergency room for fever, asthenia and weight loss. Few days before the visit, he was treated with amoxicillin/clavulanic acid for a hard and not much sore submandibular swelling, with no result. Both ultrasound and CT showed
Laura Perazzolo   +2 more
doaj   +1 more source

Lack of restricted diffusion in MR imaging as a potential differentiating tool of reactive lymph node in cat scratch disease from lymphoma or abscess

open access: yesRadiology Case Reports, 2020
Cat scratch disease is an infection caused by Bartonella Henselae with characteristic presentation of lymphadenopathy. Despite self-limited nature of the disease in most cases, it accounts for many lymph node biopsies performed since its imaging features
Youngmin Chu   +3 more
doaj   +1 more source

Cat-scratch disease manifesting as uveitis and binocular fundus nodular lesions: a case report

open access: yesBMC Ophthalmology, 2023
Background Cat-scratch disease typically presents with various ocular manifestations such as uveitis, vitritis, retinitis, retinochoroiditis, and optic neuritis. However, fundus nodular lesions was rarely reported. In our study, we reported a case of Cat-
Hao Hong   +5 more
doaj   +1 more source

Cat scratch disease

open access: yesBMJ, 2002
Unilateral lymphadenopathy of the groin is commonly seen in general practice. We report on a patient with initial features suggestive of sarcoidosis, but who, after careful history taking and further investigation, had cat scratch disease. A 29 year old woman visited her general practitioner with a painless swelling in the right groin.
Alexander, Williams   +2 more
openaire   +3 more sources

CCDC80 suppresses high‐grade serous ovarian cancer migration via negative regulation of B7‐H3

open access: yesMolecular Oncology, EarlyView.
PAX8 is a lineage‐specific master regulator of transcription in high‐grade serous ovarian cancer (HGSC) progression. We show for the first time that PAX8 facilitates proliferation and metastasis by repressing the cell autonomous tumor suppressor CCDC80 and inducing B7‐H3 expression.
Aya Saleh   +12 more
wiley   +1 more source

Loss of IGF‐1R impairs DNA‐PKcs recruitment to chromatin leading to defective end‐joining

open access: yesMolecular Oncology, EarlyView.
IGF‐1R promotes radioresistance by facilitating DNA‐PKcs recruitment to chromatin, enabling non‐homologous end‐joining (NHEJ) repair of double‐strand breaks. Inhibition or loss of IGF‐1R disrupts this recruitment to damage sites, driving compensatory reliance on microhomology‐mediated end‐joining (MMEJ) repair.
Matthew O. Ellis   +3 more
wiley   +1 more source

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