Results 201 to 210 of about 1,759,912 (296)

The impact of Hnrnpl deficiency on transcriptional patterns of developing muscle cells

FEBS Open Bio, EarlyView.
We performed nanopore whole‐transcriptome sequencing comparing RNA from Hnrnpl‐knockdown versus control C2C12 myoblasts to investigate the contributions of Hnrnpl to muscle development. Our results indicate that Hnrnpl regulates the expression of genes involved with Notch signaling and skeletal muscle, particularly splicing patterns of specific muscle ...
Hannah R. Littel, Mekala Gunasekaran, Audrey L. Daugherty, Natalya M. Wells, Johnnie Turner, Christine C. Bruels, Christina A. Pacak, Isabelle Draper, Peter B. Kang   +8 more
wiley   +1 more source

An Investigation of the Deaths in the Standard Assurance Company. By Dr. Robert Christison. - Medical Statistics of Life Assurance—Observations on the Causes of Death among the Assured of the Scottish Widows' Fund and Life Assurance Society, from 1846 to 1852. By Dr. James Begbie.

openalex   +1 more source

THE INTERNATIONAL CLASSIFICATION OF CAUSES OF SICKNESS AND DEATHS

, 1912
Jorge Le-Roy Y. Cassá
openalex   +2 more sources

Role of Biological Age in the Determination of Long-Term Cause-Specific Death Following Percutaneous Coronary Interventions. [PDF]

J Am Heart Assoc
Singh M, Friedman PA, Gulati R, El Sabbagh A, Lewis BR, Kanwar A, Raphael CE, Al-Hijji MA, Attia ZI, Behfar A, Kirkland JL.   +10 more
europepmc   +1 more source

Mitochondria‐associated membranes (MAMs): molecular organization, cellular functions, and their role in health and disease

FEBS Open Bio, EarlyView.
Mitochondria‐associated membranes (MAMs) are contact sites between the endoplasmic reticulum and mitochondria that regulate calcium signaling, lipid metabolism, autophagy, and stress responses. This review outlines their molecular organization, roles in cellular homeostasis, and how dysfunction drives neurodegeneration, metabolic disease, cancer, and ...
Viet Bui, Maryline Santerre, Natalia Shcherbik, Bassel E Sawaya   +3 more
wiley   +1 more source

Capillary leak phenotype as a major cause of death in patients with POEMS syndrome. [PDF]

Leukemia
Lee K, Kourelis T, Tschautscher M, Warsame R, Buadi F, Gertz M, Muchtar E, Dingli D, Hayman S, Go R, Hwa L, Fonder A, Gonsalves W, Hobbs M, Kyle R, Kapoor P, Leung N, Binder M, Cook J, Lin Y, Rogers M, Rajkumar SV, Kumar S, Dispenzieri A.   +23 more
europepmc   +1 more source

Production of antibacterial compounds by a Steely hybrid polyketide synthase in Dictyostelium

FEBS Open Bio, EarlyView.
By selecting appropriate production species under optimised culture conditions, new chlorinated dibenzofuran compounds, which previously could not be analysed in sufficient quantities, were identified from cellular slime mould fruiting bodies. The strong antibacterial activity exhibited by these compounds suggests a link to the ecological function of ...
Tomoaki R. Yamashita, Toyonobu Usuki, Robert R. Kay, Tamao Saito   +3 more
wiley   +1 more source

OBSERVATIONS ON THE CAUSES OF DEATH AFTER THE OPERATION OF LITHOTOMY. [PDF]

Holmes Coote
openalex   +1 more source

Quantifying years of life lost in Australia: a multiple cause of death analysis. [PDF]

Int J Epidemiol
Joshy G, Bishop K, Li H, Moran L, Gourley M, Welsh J, Korda R, Banks E, Adair T, Rao C.   +9 more
europepmc   +1 more source

Lactoferrin treatment activates acetylcholinesterase, decreasing acetylcholine levels in non‐small cell lung cancer (NSCLC) cell culture supernatants, inhibiting cell survival

FEBS Open Bio, EarlyView.
Representation of the suggested mode of action of lactoferrin (Lf) in nonsmall cell lung cancer (NSCLC) A549 cells. Lf induces activation of caspase‐3 by activating p53 and AChE leading to decreased ACh concentrations. In turn, ACh signaling leads to activation of VEGF and AKT and blocking of caspase‐3.
Stuti Goel, Caroline Wozniak, Aya Sabri, Ben Haddad, Brooke Lopo, Alvaro Cobos, Sarah Sarofim, Jeffrey Guthrie, Deborah Heyl, Hedeel Guy Evans   +9 more
wiley   +1 more source