Results 241 to 250 of about 8,976 (287)
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REVERSAL OF CENTRAL ANTICHOLINERGIC SYNDROME BY GALANTHAMINE
Survey of Anesthesiology, 1978Ten volunteers were given 2 mg scopolamine intravenously (IV) to produce substantial drowsiness and sleepiness. Galanthamine, 0.5 mg/kg IV, effectively reversed the central anticholinergic syndrome produced by scopolamine. Electroencephalographic monitoring of two subjects matched the observed changes of consciousness: scopolamine replaced the dominant
A, Baraka, S, Harik
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Reversal of Fortune: Central Nervous System Blastomycosis
The American Journal of Medicine, 2016PRESENTATION When a patient presents with simultaneous lung and brain lesions, the clinician is likely to invokemetastatic malignancy as the unifying explanation, which will usually be the correct one—but not always. In our case, a 73-year-old man originally from Bangladesh with a history of hypertension, diabetes mellitus, and curatively resected ...
Jessica Gupta +2 more
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Archives of Neurology, 1961
"Central pain" has been defined as spontaneous pain with painful over-reaction to external stimuli resulting from lesions confined to the central nervous system. 12 As Walker 15 pointed out, central pain can originate from any level of the nervous system—peripheral, spinal, bulbar, or cortical.
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"Central pain" has been defined as spontaneous pain with painful over-reaction to external stimuli resulting from lesions confined to the central nervous system. 12 As Walker 15 pointed out, central pain can originate from any level of the nervous system—peripheral, spinal, bulbar, or cortical.
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Reversal of Central Sleep Apnea Using Nasal CPAP
Chest, 1986Based on the theory that obstructive (OSA) and central (CSA) sleep apneas share common pathophysiologic mechanisms, we attempted to treat eight patients with predominantly CSA by continuous positive airway pressure (CPAP). All patients exhibited repetitive episodes of CSA and mixed sleep apneas (MSA) in the supine position with a mean duration of 23.7 +
F G, Issa, C E, Sullivan
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Reversal of synaptic vesicle docking at central synapses
Nature Neuroscience, 1999We used quantitative fluorescence imaging of vesicles labeled with membrane-soluble dyes to determine rates of undocking and spontaneous exocytosis of vesicles docked to the active zone of hippocampal synapses in culture. Individual vesicles undock about once per two minutes and spontaneously exocytose about once per eight minutes.
V N, Murthy, C F, Stevens
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Reversible central nervous system dysfunction in folate deficiency
Journal of the Neurological Sciences, 1975An epileptic patient on chronic anticonvulsant drug therapy is described, in whom anaemia and neurological abnormalities including progressive dementia, bilateral pyramidal tract signs, incontinence and ataxia developed. Vitamin B12 serum levels and absorption were normal, but serum folic acid levels were low.
E, Melamed, A, Reches, C, Hershko
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CENTRAL ANTICHOLINERGIC SYNDROME REVERSED BY TETRAHYDROAMINACRINE (THA)
Medical Journal of Australia, 1975A case of central anticholinergic syndrome after an overdose of orphenadrine citrate (Norflex), treated with tetrahydroaminacrine (THA, Tacrine) is presented. A possible mechanism for the syndrome is discussed in relation to other psychotic states, and the use of anticholinesterase agents in the treatment of toxic psychoses reviewed.
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Central structure preservation of the reversal sign
Neuroradiology, 1999We report serial changes of central structure preservation of the reversal sign in a case of child abuse. The serial CT images show that the relatively spared attenuation at the basal ganglia, thalami, and posterior fossa develops before the occurrence of transtentorial herniation. This finding makes the theory that central preservation of the reversal
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Central, naloxone-reversible antinocicepeion by diciofenae in the rat
European Journal of Pharmacology, 1990The antinociceptive effect of subcutaneously (s.c.), intracerebroventricularly (i.c.v.) or intrathecally (i.t.) administered diclofenac was studied in a series of experiments employing the tail-flick (0.01-10.0 mg/kg body weight i.p., 1-50 micrograms i.c.v., 1-10 micrograms i.t.) and hot-plate (0.01-50 mg/kg body weight i.p., 1-50 micrograms i.c.v., 1 ...
R, Björkman +3 more
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Studies on new, centrally active and reversible acetylcholinesterase inhibitors
Neurochemical Research, 1990We have synthesized the tertiary amines of pyridostigmine and neostigmine, 3-pyridinol dimethylcarbamate (norpyridostigmine) and 3-dimethylaminophenol dimethylcarbamate (norneostigmine) respectively, and we have tested their abilities to cross the blood-brain barrier and inhibit mouse brain AChE activity.
F, Arnal +5 more
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