Results 251 to 260 of about 383,227 (306)
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Postprandial cerebral infarction

Journal of Clinical Neuroscience, 2021
Some neurological diseases are accompanied by autonomic dysfunction. Postprandial hypotension (PPH) is one disorder accompanied by autonomic dysfunction. Although the major symptoms of PPH are fall and syncope, PPH is sometimes overlooked because of its non-specific symptoms, such as dizziness, nausea, and light-headedness.
Hajime, Ikenouchi   +2 more
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Perinatal cerebral infarction

Annals of Neurology, 1984
AbstractThe diagnosis of perinatal cerebral infarction, although frequently suggested clinically, has been made most commonly at postmortem examination; few infants surviving stroke are reported in the literature. We evaluated 18 infants with perinatal cerebral infarction in a recent twelve‐month interval.
L R, Ment, C C, Duncan, R A, Ehrenkranz
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Massive Cerebral Infarction

The Neurologist, 2005
Massive cerebral infarcts cause brain edema with midline shifts and impingement on vital structures producing coma and death. The mortality rate is estimated at 80% with standard medical treatment. Surgical decompression with hemicraniectomy has proved to be life saving, but the impact on functional outcomes is largely unknown. The focus of this review
Suresh, Subramaniam, Michael D, Hill
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Neonatal cerebral infarction

Seminars in Pediatric Neurology, 2000
Neonatal cerebral infarction in term infants has many possible causes, including bacterial meningitis, inherited or acquired coagulopathies, trauma, and hypoxia-ischemia. However, a specific cause often cannot be identified. Neurologic symptoms in the neonatal period are often subtle and nonspecific, even in infants with large infarctions involving an ...
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Angiography in Cerebral Infarction

Radiology, 1969
The diagnosis of cerebral infarction by angiography is based on a variety of findings, involving both anatomical and pathophysiological considerations. These findings at the time of angiography are dependent upon the type of infarction that we are dealing with (hemorrhagic versus nonhemorrhagic), its location, and the time interval between the onset of
J M, Taveras   +4 more
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CEREBRAL ISCHEMIA AND INFARCTION

Neuroimaging Clinics of North America, 1994
Cerebral infarction is most commonly related to atherosclerotic disease in the carotid and vertebrobasilar circulations. TIAs are manifestations of this same disease process and may occur before a complete infarction. The transformation of pale to hemorrhagic infarction may result from reperfusion of an infarct or from migration of an embolus.
A N, Hasso, W A, Stringer, K D, Brown
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Unilateral watershed cerebral infarcts

Neurology, 1986
We studied 51 patients with symptomatic unilateral watershed (WS) cerebral infarct on CT. In 22 patients, the infarct was between the superficial territory of the anterior and middle cerebral arteries, 20 had an infarct between the superficial territory of the middle and posterior cerebral arteries, and 9 had an infarct between the superficial and deep
J, Bogousslavsky, F, Regli
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Extensive hemispheric cerebral infarction

Journal of Stroke and Cerebrovascular Diseases, 1998
Patients with extensive hemispheric cerebral infarction have a high incidence of mortality and serious morbidity. Because of their poor prognosis, they warrant attention; however, in acute stroke therapy trials they do not appear to benefit from treatment.
C R, Levi   +3 more
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Venous cerebral infarction

Surgical Neurology, 1981
K S, Kim, P E, Weinberg
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