Results 351 to 360 of about 1,282,519 (402)
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Hemichannels in Cerebral Ischemia
Current Molecular Medicine, 2009Hemichannels are transmembrane channels that represent the functional subunits of gap junctions. Each hemichannel is composed of a connexin or pannexin hexamer and, after being transported to the membrane, remains unpaired until it is incorporated in a gap junction. Several studies have already provided evidence that gap junction-mediated intercellular
Markus Schwaninger +2 more
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2011
Rodent models of focal cerebral ischemia have been extremely useful in elucidating pathomechanisms of human stroke. Most commonly, a monofilament is advanced through the internal carotid artery of rodents to occlude the origin of the middle cerebral artery thus leading to critical ischemia in the corresponding vascular territory.
Guido Stoll +3 more
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Rodent models of focal cerebral ischemia have been extremely useful in elucidating pathomechanisms of human stroke. Most commonly, a monofilament is advanced through the internal carotid artery of rodents to occlude the origin of the middle cerebral artery thus leading to critical ischemia in the corresponding vascular territory.
Guido Stoll +3 more
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CEREBRAL ISCHEMIA AND INFARCTION
Neuroimaging Clinics of North America, 1994Cerebral infarction is most commonly related to atherosclerotic disease in the carotid and vertebrobasilar circulations. TIAs are manifestations of this same disease process and may occur before a complete infarction. The transformation of pale to hemorrhagic infarction may result from reperfusion of an infarct or from migration of an embolus.
A N, Hasso, W A, Stringer, K D, Brown
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Cell Death Mechanisms in Cerebral Ischemia–Reperfusion Injury
Neurochemical Research, 2022Qian-ru Zhang +4 more
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Focal Cerebral Ischemia and Migraine
Cephalalgia, 1985Consistent literature data on the cerebrovascular risk in migraineous patients are lacking. Available preliminary clinical data (Italian Cooperative Cross-Sectional Case-Control Study) suggest that migraine can hardly be considered a relevant pathogenetic associated risk factor of focal cerebral ischemia.
PRENCIPE M +3 more
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Therapeutics for Focal Cerebral Ischemia
Neurosurgery, 1980A review of the therapeutic modalities available for the treatment of focal cerebral ischemia is presented. The theoretical aspects and present practical applications of each treatment are discussed. The possibilities for future research and for the implementation of new modalities are indicated.
Warren R. Selman, Robert F. Spetzler
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Electrophysiology of cerebral ischemia
Neuropharmacology, 2008Organized brain activity requires the coordinated firing of vast numbers of nerve cells. To maintain this, all these cells must be adequately polarized, their axons capable of conducting action potentials and releasing transmitters at an even greater numbers of synapses. Hence the often dire consequences of any interruption in the normal supply of O(2)
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Polyamines and cerebral ischemia
1998It has been well established that alterations in polyamine metabolism are associated with animal models of global ischemia. Recently, this has been extended to include models of focal ischemia and traumatic brain injury. There is much evidence to support the idea that polyamines may play a multifaceted detrimental role following ischemia reperfusion ...
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Total Cerebral Ischemia in the Dog
Archives of Surgery, 1962In a previous report1we described a simple method of selectively cooling the brain as a possible method of allowing prolonged interruption of the cerebral circulation. It was apparent then that exact knowledge of the time tolerance of the brain to ischemia at various cerebral temperatures was needed if this technique was going to be applied clinically.
Robert J. Boyd, John E. Connolly
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Effects of cerebral ischemia in mice deficient in neuronal nitric oxide synthase.
Science, 1994The proposal that nitric oxide (NO) or its reactant products mediate toxicity in brain remains controversial in part because of the use of nonselective agents that block NO formation in neuronal, glial, and vascular compartments. In mutant mice deficient
Zhihong Huang +5 more
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