Results 21 to 30 of about 59,976 (260)

Papilledema and venous stasis in patients with cerebral venous and sinus thrombosis

open access: yesBMC Neurology, 2023
Background Cerebral venous and sinus thrombosis (CVST) can cause increased intracranial pressure, often leading to papilledema. In this study, we investigated the association between papilledema and venous stasis on susceptibility weighted imaging (SWI ...
Min-Gyu Park   +4 more
doaj   +1 more source

Does massive cerebral venous thrombosis mean poor outcome? [PDF]

open access: yesMedicinski Glasnik, 2011
A 21-year-old woman taking oral contraceptives presented with headaches, nausea, vomiting and somnolence. The next day she had a generalized tonic-clonic seizure after which her neurologicalcondition deteriorated.
Petar Slankamenac   +7 more
doaj  

Hyperhomocysteinemia in cerebral vein thrombosis

open access: yesBlood, 2003
Abstract High plasma levels of total homocysteine (tHcy) are a risk factor for deep vein thrombosis. Because no information on the relationship between cerebral vein thrombosis and hyperhomocysteinemia is available, a case–control study of 121 patients with a first episode of cerebral vein thrombosis and 242 healthy control subjects was ...
I. Martinelli   +4 more
openaire   +3 more sources

The Editor’s Corner

open access: yesVeins and Lymphatics, 2013
Cerebral Venous Return is a novel, exciting field of research. As a pioneer, I would say that when I began investigating the intra and extra cranial cerebral veins, very few data were available something about 10-12 years ago...
Paolo Zamboni
doaj   +1 more source

EDNRB‐dependent endothelin signaling reduces proliferation and promotes proneural‐to‐mesenchymal transition in gliomas

open access: yesMolecular Oncology, EarlyView.
Glioma cells mainly express the endothelin receptor EDNRB, while EDNRA is restricted to a perivascular tumor subpopulation. Endothelin signaling reduces glioma cell proliferation while promoting migration and a proneural‐to‐mesenchymal transition associated with poor prognosis. This pathway activates Ca2+, K+, ERK, and STAT3 signalings and is regulated
Donovan Pineau   +36 more
wiley   +1 more source

SINUS DURAE MATRIS AND Vv. CEREBRI IN MULE (Equus hinnus) [PDF]

open access: yesAssiut Veterinary Medical Journal, 1985
The examined Sinus durae matris of the mule include a dorsal and ventral system. The dorsal system comprises Sinus sagitalis dorsalis, Sinus rectus, Sinus transversus and Sinus occipitalis dorsalis, while the ventral system includes Sinus cavernosus ...
A.K. AHMED, M.A. METWALLY, A. HIFNY
doaj   +1 more source

Engineered extracellular vesicles enriched with the miR‐214/199a cluster enhance the efficacy of chemotherapy in ovarian cancer

open access: yesMolecular Oncology, EarlyView.
Loss of the miR‐214/199a cluster is associated with recurrence in ovarian cancer. Engineered small extracellular vesicles (m214‐sEVs) elevate miR‐214‐3p/miR‐199a‐5p in tumor cells, suppress β‐catenin, TLR4, and YKT6 signaling, reprogram tumor‐derived sEV cargo, reduce chemoresistance and migration, and enhance carboplatin efficacy and survival in ...
Weida Wang   +12 more
wiley   +1 more source

Measurement of cerebral venous oxygenation with quantitative susceptibility mapping after subarachnoid hemorrhage: A pilot study

open access: yesJournal of Central Nervous System Disease
Objective We measured cerebral venous oxygenation after aneurysmal subarachnoid hemorrhage (aSAH) using quantitative susceptibility mapping (QSM) to explore its relationship with cognitive function. Methods Twenty participants, including 10 patients with
Meng Qi   +6 more
doaj   +1 more source

Clinical and radiological features of cerebral venous thrombosis by the example of clinical cases

open access: yesКлинический разбор в общей медицине, 2023
Venous blood accounts for more than 80% of cerebral blood flow. At the same time, the incidence of cerebral venous thrombosis in patients with ischaemic stroke is only 3%.
Oleg V. Zolotarev   +3 more
doaj   +1 more source

Functional and Structural Evidence of Neurofluid Circuit Aberrations in Huntington Disease

open access: yesAnnals of Clinical and Translational Neurology, EarlyView.
ABSTRACT Objective Disrupted neurofluid regulation may contribute to neurodegeneration in Huntington disease (HD). Because neurofluid pathways influence waste clearance, inflammation, and the distribution of central nervous system (CNS)–delivered therapeutics, understanding their dysfunction is increasingly important as targeted treatments emerge.
Kilian Hett   +8 more
wiley   +1 more source

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