Chlamydia psittaci inhibits apoptosis of human neutrophils by activating P2X7 receptor expression
This study tested the hypothesis that Chlamydia psittaci (C. psittaci) survives and multiplies in human neutrophils by activating P2X7, a nonselective cationic channel receptor expressed constitutively on the surface of these cells.
Zhangping He+11 more
doaj
Isolation of Chlamydia psittaci from a patient with interstitial keratitis and uveitis associated with otological and cardiovascular lesions. [PDF]
S Darougar+4 more
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Loss of Inorganic Ions from Host Cells Infected with Chlamydia psittaci [PDF]
George T. Chang, James W. Moulder
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Toxicity of Low and Moderate Multiplicities of Chlamydia psittaci for Mouse Fibroblasts (L Cells) [PDF]
Kevin R. Kellogg+2 more
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Prevalence of Chlamydophila psittaci in wild birds—potential risk for domestic poultry, pet birds, and public health? [PDF]
To determine the prevalence of Chlamydophila psittaci in wild birds, cloacal swabs from 527 songbirds, 442 waterfowl, 84 feral pigeons, and 38 cormorants were examined by Chlamydiaceae-specific real-time polymerase chain reaction (PCR) and ArrayTube ...
Borel, Nicole+4 more
core
Psittacosis is a infectious disease caused by Chlamydia psittaci (C. psittaci), which presents as pneumonia in humans. The diagnosis of psittacosis is challenging, however, Metagenomic next-generation sequencing (MNGS) is very efficient.
Chunhua Luo+4 more
doaj
Hemagglutinin in Cell Walls ofChlamydia psittaci [PDF]
Akira Tamura, G. P. Manire
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Identificación de Chlamydophila psittaci en aves psitácidas del hogar de paso de fauna silvestre “La María”, Pereira [PDF]
Las psitácidas son una familia de aves de características morfológicas específicas, que abarcan un gran número de especies distribuidas en su mayor parte en América; la psitacosis es una enfermedad infectocontagiosa que este tipo de aves padecen, es ...
Rivera Osorio, Sebastián
core
Inhibition of Deoxyribonucleic Acid Synthesis in Synchronized Populations of L Cells Infected with Chlamydia psittaci [PDF]
Ilse I. E. Tribby, James W. Moulder
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