Results 161 to 170 of about 272,468 (312)
664 Treatment with erythropoietin improves left ventricular systolic performance and mitral regurgitation in patients with heart failure and chronic kidney disease [PDF]
Bernard Cosyns+4 more
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sEVs have a critical role in orchestrating interorgan crosstalk and mediating exercise‐induced therapeutic effects. Lin et al. demonstrates that sEVs miR‐17/20a‐5p mediates the muscle‐brain crosstalk and emphasizes the central role of mTOR signaling in executing molecular programs that can protect brain health in response to exercise. Abstract Physical
Huawei Lin+21 more
wiley +1 more source
Multimorbidity Due to Diabetes Mellitus and Chronic Kidney Disease and Outcomes in Chronic Heart Failure [PDF]
O. James Ekundayo+5 more
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This study constructs a stacked multimodal machine learning model for nonalcoholic fattly liver disease (NAFLD) by integrating genetic and clinical features, and synthesize an in‐silico quantitative marker (ISNLD) that enables personalized risk stratification for intrahepatic and extrahepatic outcomes of high‐risk individuals of NAFLD.
Lushan Xiao+14 more
wiley +1 more source
MORPHOPATHOLOGICAL STUDY ON THE EXTIRPATED KIDNEYS IN DIALYZED PATIENTS WITH CHRONIC RENAL FAILURE
Junzo Nakanishi
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Pan-ureteric transitional cell carcinoma in a patient of autosomal dominant polycystic kidney disease with chronic renal failure [PDF]
Sunil Kumar+2 more
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Schematic diagram showing the potential mechanism of bigelovin on the activation of NLRP3 inflammasome Bigelovin may inhibit activated protein C kinase 1 (RACK1) by directly binding with cys168 of RACK1. Bigelovin thus prevents oligomerization of NLRP3 (NLRP3 active conformation) and subsequent assembly of NLRP3 inflammasome, blocking the activation of
Jian Cui+17 more
wiley +1 more source
BONE FORMATION IN THE BILATERAL KIDNEYS OF A CHRONIC RENAL FAILURE PATIENT A Case Report
Seiji Kudoh+4 more
openalex +2 more sources
Recombinant Human Epo Treatment: Beneficial in Chronic Kidney Disease, Chronic Heart Failure, or Both? [PDF]
Anne M.S. Belonje+2 more
openalex +1 more source
This study identifies alnustone, a natural compound from Alpinia katsumadai, as a potent therapeutic agent for MASLD and MASH. Alnustone enhances mitochondrial fatty acid β‐oxidation by directly targeting calmodulin, improving liver steatosis, fibrosis, and insulin resistance in vivo.
Shourui Hu+13 more
wiley +1 more source