Catheter removal and outcomes of multidrug-resistant central-line-associated bloodstream infection [PDF]
Burnham, Jason P. +2 more
core +1 more source
Unilateral Acroangiodermatitis: From Histopathologic Confirmation to Treatment with PDL. [PDF]
Aparício Martins A +2 more
europepmc +1 more source
Sabotaged Integral HSC Heterogeneity Underlies Essential Thrombocythemia Development
Single‐cell RNA sequencing (scRNA‐seq) maps how distinct driver mutations remodel hematopoietic stem cell (HSC) programs across essential thrombocythemia (ET). Comparative analysis uncovers both shared and subtype‐specific molecular signatures, identifies a triple‐negative (TN)‐associated HSC population enriched with malignant traits, and reveals the ...
Jingyuan Tong +21 more
wiley +1 more source
Pathophysiological mechanisms of chronic venous disease and their role in C0s clinical class
Andrew Nicolaides, Eliete Bouskela
openalex +1 more source
Impact of hemodynamic doppler ultrasound parameters on patency and post-thrombotic syndrome in patients with iliofemoral venous stents. [PDF]
Skaff Y +8 more
europepmc +1 more source
CircSMEK1 Suppresses HCC via the hnRNPK‐IGF2‐AKT Axis: A Diagnostic Biomarker and Therapeutic Target
CircSMEK1 is downregulated in MASH/HCC and predicts poor prognosis. It suppresses tumor progression by promoting hnRNPK ubiquitination and inhibiting the IGF2/PI3K/AKT axis, while its loss activates immunosuppressive cancer‐associated fibroblasts. Serum circSMEK1 serves as a non‐invasive diagnostic biomarker, and its restoration potently inhibits HCC ...
Peilan Guo +15 more
wiley +1 more source
The burden of chronic venous diseases is unknown [PDF]
openaire +2 more sources
Therapeutic Outcomes of Graduated and Progressive Compression in Chronic Venous Disorders: A Meta-Analysis. [PDF]
Hassan Awaji HH +10 more
europepmc +1 more source
This work demonstrates that endothelial EphA4 limits Tie2 activation and curbs leptomeningeal collateral expansion after ischemic stroke. Pharmacological activation of Tie2 by an Ang‐1 mimetic or genetic loss of EphA4 stimulates collateral vessel growth, improves cerebral perfusion, and enhances neurological recovery.
Alexandra M. Kaloss +9 more
wiley +1 more source

