Results 91 to 100 of about 571 (131)
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Neurology, 2002
Chronic inflammatory demyelinating polyneuropathy (CIDP) is an autoimmune disease that targets the myelin sheaths of peripheral nerves. In clinical practice the diagnosis is often difficult to make because of the clinical heterogeneity of the disease, its multifocality and predilection for proximal nerve segments, and the limitations of our ...
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Chronic inflammatory demyelinating polyneuropathy (CIDP) is an autoimmune disease that targets the myelin sheaths of peripheral nerves. In clinical practice the diagnosis is often difficult to make because of the clinical heterogeneity of the disease, its multifocality and predilection for proximal nerve segments, and the limitations of our ...
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MR neurography for the evaluation of CIDP
Muscle & Nerve, 2016ABSTRACT Introduction : To visualize peripheral nerves in patients with chronic inflammatory demyelinating polyneuropathy (CIDP), we used MR imaging. We also quantified the volumes of the brachial and lumbar plexus and their nerve roots.
Tomomasa, Ishikawa +12 more
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Chronische inflammatorische demyelinisierende Polyradikuloneuropathie (CIDP)
Neurologie up2date, 2023Die chronische inflammatorische demyelinisierende Polyradikuloneuropathie (CIDP) ist die häufigste chronische Autoimmunerkrankung des peripheren Nervensystems. Sie ist häufig schwierig zu diagnostizieren. Die CIDP ist mittlerweile mittels verschiedener Immuntherapeutika gut behandelbar.
Barbara Kaulen +2 more
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Neurology, 2003
Current knowledge about the pathogenic mechanisms involved in chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) supports an autoimmune etiology. Some of the cell and humorally mediated immune responses that contribute to the development of CIDP resemble those implicated in multiple sclerosis (MS).
Klaus V, Toyka, Ralf, Gold
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Current knowledge about the pathogenic mechanisms involved in chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) supports an autoimmune etiology. Some of the cell and humorally mediated immune responses that contribute to the development of CIDP resemble those implicated in multiple sclerosis (MS).
Klaus V, Toyka, Ralf, Gold
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Neurology, 2003
This article reviews the efficacy and tolerability of currently available therapies, including intravenous immunoglobulin (IVIg), corticosteroids, and plasma exchange (PE), for treatment of chronic inflammatory demyelinating polyradiculoneuropathy (CIDP). Data show that current therapies are effective in approximately two-thirds of patients.
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This article reviews the efficacy and tolerability of currently available therapies, including intravenous immunoglobulin (IVIg), corticosteroids, and plasma exchange (PE), for treatment of chronic inflammatory demyelinating polyradiculoneuropathy (CIDP). Data show that current therapies are effective in approximately two-thirds of patients.
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Electrophysiologic correlations with clinical outcomes in CIDP
Muscle & Nerve, 2010AbstractData are lacking on correlations between changes in nerve conduction (NC) studies and treatment response in chronic inflammatory demyelinating polyneuropathy (CIDP). This report examined data from a randomized, double‐blind trial of immune globulin intravenous, 10% caprylate/chromatography purified (IGIV‐C [Gamunex]; n = 59) versus placebo (n =
Bril, V +58 more
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Treatment of Guillain‐Barré syndrome and CIDP
Journal of the Peripheral Nervous System, 2005Abstract Guillain‐Barré syndrome (GBS) and chronic inflammatory demyelinating poly‐(radiculo)neuropathy (CIDP) are immune‐mediated disorders with a variable duration of progression and a range in severity of weakness. Infections can trigger GBS and exacerbate CIDP.
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Neurology, 2010
One of the challenges faced by neurologists seeing patients with neuromuscular disease is distinguishing one form of immune-mediated neuropathy from another.1–3 This is more than an academic question because diagnosing a patient with Guillain-Barre syndrome vs acute-onset chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) has implications
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One of the challenges faced by neurologists seeing patients with neuromuscular disease is distinguishing one form of immune-mediated neuropathy from another.1–3 This is more than an academic question because diagnosing a patient with Guillain-Barre syndrome vs acute-onset chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) has implications
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CIDP PRESENTING WITH RADICULOPATHY
Journal of Neurology, Neurosurgery & Psychiatry, 2012A 62-year-old gentleman presented in 2009 with pain, numbness and tingling in the right hand and arm. Neurological examination revealed reduced light touch and pain sensation in the C8 dermatome. Significant past medical history included neck trauma in 1989, managed by C4/5 and C5/6 cervical decompression. MRI revealed only mild alkylotic changes at C4/
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