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Management of CML-blast crisis
Best Practice & Research Clinical Haematology, 2016Tyrosine kinase inhibitors (TKI) have moderately improved survival in BC, but a median survival of less than 1 year is still unsatisfactory. This article reviews the various tests required for diagnosis of BC, features at diagnosis, treatment modalities (intensive chemotherapy, TKI, allo-SCT and a selection of investigational agents), options of ...
Susanne Saußele +3 more
openaire +3 more sources
Blood, 2004
Comment on Pelletier et al, page [2163][1] Pelletier and colleagues report that the lack of the adhesion molecules P-selectin and ICAM-1 accelerates the development of BCR/ABL-induced CML-like disease in mice. The data have important implications for understanding as well as treating CML.
openaire +2 more sources
Comment on Pelletier et al, page [2163][1] Pelletier and colleagues report that the lack of the adhesion molecules P-selectin and ICAM-1 accelerates the development of BCR/ABL-induced CML-like disease in mice. The data have important implications for understanding as well as treating CML.
openaire +2 more sources
Stem cell persistence in CML is mediated by extrinsically activated JAK1-STAT3 signaling
Leukemia, 2019Maja Kim Kuepper +13 more
semanticscholar +1 more source
2007
Abstract : The purpose of this proposal is to understand the molecular pathways regulating Bcr-Abl positive CML cells. We demonstrated that the transcription factor, CREB, is highly expressed in K562 cells and cells from patients with chronic phase CML.
openaire +2 more sources
Abstract : The purpose of this proposal is to understand the molecular pathways regulating Bcr-Abl positive CML cells. We demonstrated that the transcription factor, CREB, is highly expressed in K562 cells and cells from patients with chronic phase CML.
openaire +2 more sources