Radioresistance arises partly from tumor cells evading cuproptosis via unknown defenses. This study reveals that the PRMT5‐VPS34 axis acts as a radiation‐activated anticuproptotic mechanism: PRMT5 methylates VPS34 at Arg174, recruiting USP10 to remove K48‐linked ubiquitination and prevent degradation.
Wei Chen +17 more
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Submucosal metastatic leiomyosarcoma of the colon presenting with intussusception and lower gastrointestinal bleeding: a case report. [PDF]
Li H, Yu Z, Ye M, Jin Q.
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Surgical approaches to esophageal cancer gastrointestinal reconstruction: a systematic review of jejunum, colon, and ileocolonic interposition with comparative outcome analysis. [PDF]
Liu X, Dai J, Yin M, Zhang R, Song Y.
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Synchronous Primary Adenocarcinomas of the Hepatic and Splenic Flexures Presenting as Acute Large Bowel Obstruction in a 38-Year-Old Man: A Case Report. [PDF]
Gill J, Brady HW, Mbachi C.
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Surgical Colonic Myeloid Sarcoma Following Bone Marrow Transplantation in a Patient With Acute Myeloid Leukemia With Myelodysplasia-Related Changes: A Case Report. [PDF]
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Management of two extremely aggressive signet ring cell colon carcinoma: a case series. [PDF]
Dalir N, Parker GS, Masia RA, Torres P.
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Initial management strategies for right-sided malignant colonic obstruction: a systematic review and bayesian network meta-analysis. [PDF]
Kitaguchi D +8 more
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Incidence and Survival of Gastro-Entero-Pancreatic Neuroendocrine Neoplasms in the Contemporary Era.
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Perforation of colonic neoplasms
International Journal of Colorectal Disease, 1987Colonic perforation is the second most common complication of colonic neoplasms and is associated with an elevated morbidity and mortality. We undertook a two-centre retrospective analysis of 378 colonic neoplasms seen from 1978 to 1985. Thirty-six patients (9.5%) presented with a perforated colonic carcinoma.
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