Results 281 to 290 of about 1,790,196 (391)
NEW METHODS OF PYLOROPLASTY FOR CONGENITAL PYLORIC STENOSIS [PDF]
Alfred A. Strauss
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Metabolic alterations in human pulmonary artery smooth muscle cells treated with PDGF‐BB
Metabolic abnormalities are considered to play a key regulatory role in vascular remodeling of pulmonary arterial hypertension. We analyzed the metabolome in the culture supernatants of human pulmonary artery smooth muscle cells (PASMC) during the malignant proliferation phenotype transition via a targeted metabolomics method. Significant and extensive
Meng‐Jie Zhang+9 more
wiley +1 more source
Large bowel obstruction in an adolescent caused by dolichocolon and a fibrotic band of Toldt: A rare case report. [PDF]
Ibrahim R+3 more
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Neutrophil‐to‐lymphocyte ratio (NLR) and monocyte‐to‐lymphocyte ratio (MLR) were investigated as potential markers. A total of 3545 subjects were included in the analysis retrospectively. Adult men and women with impaired glucose metabolism were assessed. NLR and MLR may help assess inflammation in individuals with impaired glucose metabolism. Abstract
Ayed A. Dera+11 more
wiley +1 more source
First case report of a unique combination of congenital limb and skeletal anomalies mimicking VACTERL and Gollop-Wolfgang syndromes. [PDF]
Elewee A+5 more
europepmc +1 more source
Severe congenital neutropenias
J. Skokowa+4 more
semanticscholar +1 more source
Strategies for optimization of hypoglycemia rat models
This review focuses on rat models for studying the short‐term and long‐term effects of mild and severe hypoglycemia. We explored the physiological mechanisms to understand the consequences of hypoglycemia in rat experimental models. This study sheds light on uncovering the therapeutic potential for hypoglycemic patients and its efficacy in mitigating ...
Lee Yeong Zher+4 more
wiley +1 more source
Congenital Heart Disease in the General Population: Changing Prevalence and Age Distribution
A. Marelli+4 more
semanticscholar +1 more source
Deleting Col6a3 exon 3 by CRISPR in mice results in centralized nuclei consistent with a myopathy phenotype mimicking collagen VI‐associated human disease Abstract Background Most mutations in the COL6A3 gene lead to collagen VI‐related myopathies. This is due to a reduced expression or mislocalization of the COL6A3 protein.
Michel ElChoueiry+12 more
wiley +1 more source