Results 151 to 160 of about 49,984 (235)

Predictive value of the uric acid-to-albumin ratio for contrast-induced nephropathy after percutaneous coronary intervention and construction of a clinical prediction model in patients with coronary artery disease. [PDF]

Front Cardiovasc Med
Zhu Y, Liu Q, Deng Y, Chen J, Chen Y, Chen B.   +5 more
europepmc   +1 more source

Investigation of the Efficacy of Orientin in Streptozotocin‐Induced Type 1 Diabetes Model and Pancreatic Beta‐TC6 Cells

Journal of Biochemical and Molecular Toxicology, Volume 40, Issue 8, August 2026.
Orientin, a natural flavonoid, protects against streptozotocin‐induced pancreatic β‐cell damage by suppressing NF‐κB–mediated inflammation, restoring antioxidant defense (SOD, CAT, GSH), and inhibiting apoptotic (Bax/Bcl‐2, Caspase‐3/9) and pyroptotic (Caspase‐1, IL‐1β) cell death pathways in both an in vivo type 1 diabetes mouse model and Beta‐TC6 ...
Bengul Ozdemir Sarikaya, Seyit Ali Bingol   +1 more
wiley   +1 more source

Bright kidney: a sign of contrast induced nephropathy after percutaneous coronary intervention. [PDF]

Oxf Med Case Reports
Omote K, Aikawa T, Sunaga D, Funayama N.
europepmc   +1 more source

Dexpanthenol Attenuates Methotrexate‐Induced Nephrotoxicity Through Modulation of NF‐κB–Mediated Inflammation and SIRT1/PGC‐1α–NRF2/HO‐1–Associated Oxidative Stress Pathways

Journal of Biochemical and Molecular Toxicology, Volume 40, Issue 8, August 2026.
Graphical abstract depicting the protective role of DEX against MTX‐induced nephrotoxicity. MTX exposure activates NF‐κB, leading to upregulation of TNF‐α and downstream apoptotic mediator Cas‐3, which collectively drive inflammation, apoptosis, and renal tissue damage. Simultaneously, MTX suppresses protective regulators SIRT1, PGC‐1α, NRF2, and HO‐1,
Atila Altuntas, Halil Asci, Esma Selcuk, Huzeyfe Karaosman, Osman Aydin, Ilter Ilhan, Sefa Alperen Ozturk, Ozlem Ozmen   +7 more
wiley   +1 more source

Efficacy of Coronary Sinus Aspiration in Reducing Contrast-Induced Nephropathy in High-Risk Patients. [PDF]

JACC Adv
Verma S, Ansari AH, Kumar R, Jain A, Aggarwal A, Bansal S, Isser HS.   +6 more
europepmc   +1 more source

Isobavachin Attenuates Renal and Cardiac Injury in Chronic Hyperuricemia via Modulation of NF‐κB and NLRP3 Signaling Pathways

Journal of Biochemical and Molecular Toxicology, Volume 40, Issue 8, August 2026.
Isobavachin alleviates HUA‐induced oxidative stress, inflammation, and fibrosis in the kidney and heart, potentially through its dual role in promoting UA excretion and inhibiting NF‐κB signaling and NLRP3 inflammasome activation. ABSTRACT Hyperuricaemia (HUA) is a recognized risk factor for cardiovascular and renal diseases.
Yongmei Li, Yuexin Xu, Wenjie Ye, Zeqi Yu, Zhenkun Wu, Xinyi Liu, Xuhong Huang, Jianxin Pang, Zean Zhao, Ting Wu   +9 more
wiley   +1 more source

The predictive value of AGEF score for contrast-induced nephropathy in patients with lower extremity peripheral artery disease. [PDF]

BMC Cardiovasc Disord
Ilis D, Arslan A, Artac I, Karakayali M, Omar T, Demir O, Guzel E, Kertmen O, Karabag Y, Rencuzogullari I.   +9 more
europepmc   +1 more source

SIRT Family: Biological Functions and Therapeutic Targets

MedComm, Volume 7, Issue 8, August 2026.
SIRT1–SIRT7 networks from transgenic mice to human‑relevant therapeutic targets. SIRT1–SIRT7 form an isoform‑, organ‑, and disease‑specific regulatory network. Transgenic Sirt1–7 mouse models define central regulatory SIRTs (SIRT1, SIRT3, SIRT6), context‑dependent modifiers (SIRT2, SIRT4, SIRT5, SIRT7), and their key mechanisms and target organs. These
Jia‐Yi Wang, Feng‐Li Jiang, Fang‐Yuan Zhang, Dong‐Hui Huang, Xiao‐Ying Li, Song Gao, Hua You, Qi‐Jun Wu, Huan‐Huan Chen, Ting‐Ting Gong   +9 more
wiley   +1 more source

Correction to "Calorie Restriction Protects against Contrast-Induced Nephropathy via SIRT1/GPX4 Activation". [PDF]

Oxid Med Cell Longev
europepmc   +1 more source

The Incidence of Contrast-Induced Nephropathy Following Computed Tomography and Associated Risk Factors. [PDF]

Radiol Res Pract
Mosaddegh R, Mohammadi F, Valipour AM, Hosseini SM, Naghshbandi M, Yarahmadi M, Faradonbeh NA.   +6 more
europepmc   +1 more source