Results 101 to 110 of about 53,155 (138)
Diagnostic Value of Sirtuin-1 in Predicting Contrast-Induced Nephropathy After Percutaneous Coronary Intervention. [PDF]
Ardic M, Gul CB.
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The relationship between the Fibrosis-4 (FIB-4) index, occurrence of contrast-induced nephropathy, and prognosis after percutaneous coronary intervention in acute coronary syndromes. [PDF]
Sielski J +11 more
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Reducing Contrast-Induced Nephropathy Risk in a Murine Model: Role of Avanafil and Vardenafil in Modulating Oxidant/Antioxidant Balance. [PDF]
Mavridis C +5 more
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Contrast-Induced Nephropathy [PDF]
A 48-year-old man presents to the Emergency Department and complains of new onset of chest pain with exertion. He has a history of tobacco use, hypercholesterolemia, type 2 diabetes mellitus, and chronic renal disease (baseline serum creatinine concentration [SCr] 1.7 mg/dL; estimated glomerular function [eGFR] 47 mL/min per 1.73m2).
Vasilios G. Athyros +2 more
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European Radiology Supplements, 2005
How contrast medium-induced nephropathy (CIN) comes about is not fully understood, although CIN constitutes a leading cause of renal failure. Here, a short review of clinical trials and a more thorough outline of mechanisms thought to cause CIN are outlined. Osmolality is only one of several physicochemical properties of contrast media (CM).
Peter A, McCullough, Sandeep S, Soman
openaire +4 more sources
How contrast medium-induced nephropathy (CIN) comes about is not fully understood, although CIN constitutes a leading cause of renal failure. Here, a short review of clinical trials and a more thorough outline of mechanisms thought to cause CIN are outlined. Osmolality is only one of several physicochemical properties of contrast media (CM).
Peter A, McCullough, Sandeep S, Soman
openaire +4 more sources
The Prevention of Contrast-Induced Nephropathy
Annals of Pharmacotherapy, 2014Objective: Contrast-induced nephropathy (CIN) is a complication which may develop after exposure to iodinated contrast media. The resulting acute kidney injury (AKI) is associated with an increase in both short- and long-term morbidity and mortality, increased hospital length of stay, and greater health care costs.
Daniel E Hilleman +1 more
exaly +3 more sources
Internal and Emergency Medicine, 2012
Radiological procedures utilizing intravascular iodinated contrast media are being widely applied for both diagnostic and therapeutic purposes and represent one of the main causes of contrast-induced nephropathy (CIN) and hospital-acquired renal failure.
Giancarlo, Marenzi +3 more
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Radiological procedures utilizing intravascular iodinated contrast media are being widely applied for both diagnostic and therapeutic purposes and represent one of the main causes of contrast-induced nephropathy (CIN) and hospital-acquired renal failure.
Giancarlo, Marenzi +3 more
openaire +2 more sources
Pathophysiology of Contrast-Induced Nephropathy
The American Journal of Cardiology, 2006Contrast-induced nephropathy (CIN) is the third leading cause of acute kidney injury in hospitalized patients and is associated with significant patient morbidity. The pathogenesis of CIN is complex and not fully understood, but iodinated contrast agents induce intense and prolonged vasoconstriction at the corticomedullary junction of the kidney ...
Tumlin, J +6 more
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Pathophysiology of contrast-induced nephropathy
International Journal of Cardiology, 2012Contrast media induce various factors that may increase vasoconstriction and decrease vasodilatation in the renal medulla, leading to hypoxia and acute tubular necrosis known as contrast-induced nephropathy (CIN) that tends to occur in diabetics and patients with preexisting renal insufficiency.
Philip Ching Yat, Wong +3 more
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Contrast-induced nephropathy: a review
Cardiovascular Revascularization Medicine, 2005Contrast-induced nephropathy (CIN) is one of the leading causes of renal impairment in the United States and the third cause of hospital-acquired renal failure. Reduction in the incidence of CIN can lead to a decrease in the morbidity, mortality, and length of hospital stay.
Mohammad, Sanaei-Ardekani +3 more
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