Results 301 to 310 of about 859,972 (336)
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Preventing Coronary Restenosis and Complications
New England Journal of Medicine, 1997Interventions aimed at opening occluded coronary arteries continue to evolve. Two fundamental problems that limit the clinical efficacy of coronary interventions are restenosis after coronary angio...
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The Porcine Model of Coronary Restenosis
2001Human coronary restenosis remains an elusive problem, and a major limitation of all percutaneous interventional coronary revascularization procedures, despite intracoronary stenting (1–9). Restenosis has recently gained even greater importance, since trials comparing PTCA with coronary bypass surgery (BARI, EAST, CABRI) suggest that angioplasty is ...
David R. HolmesJr.+2 more
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Circulation, 1996
BACKGROUND Neointimal formation causes restenosis after intracoronary stent placement. Endovascular radiation delivered via a stent has been shown to reduce neointimal formation after placement in porcine and rabbit iliac arteries.
A. Carter+8 more
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BACKGROUND Neointimal formation causes restenosis after intracoronary stent placement. Endovascular radiation delivered via a stent has been shown to reduce neointimal formation after placement in porcine and rabbit iliac arteries.
A. Carter+8 more
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Treatment options for coronary stent restenosis
Comprehensive Therapy, 1999Treatment of in-stent restenosis with balloon angioplasty alone is adequate for focal lesions but is associated with a 50% recurrence rate for diffuse lesions. For diffuse in-stent restenosis, debulking with atherectomy or laser can significantly reduce the recurrence rate.
David J. Cohen, Harold L. Dauerman
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Circulation, 1996
BACKGROUND Clinical restenosis after balloon angioplasty can be categorized by use of dichotomous terms based on the presence or absence of recurrent myocardial ischemia.
K. Lehmann, R. Melkert, P. Serruys
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BACKGROUND Clinical restenosis after balloon angioplasty can be categorized by use of dichotomous terms based on the presence or absence of recurrent myocardial ischemia.
K. Lehmann, R. Melkert, P. Serruys
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Management of restenosis after coronary intervention
American Heart Journal, 1996Coronary restenosis has proven to be the "Achilles heel" of percutaneous coronary interventions, frequently leading to repeated procedures. The pathogenesis of restenosis can be divided into four phases: early elasic recoil (hours to days), mural thrombus formation (hours to days), neointimal proliferation and extracellular matrix formation (weeks ...
Valentin Fuster, George Dangas
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Coronary restenosis: Evaluation of a restenosis injury index in a swine model
American Heart Journal, 1993To investigate the mechanisms of restenosis and detect useful interventions to prevent it, reliable quantitative measurements must be evaluated. Coronary arteries of domestic and minipigs (n = 18) were mechanically injured by balloon overstretching and killed at different intervals (2 to 25 weeks) after quantitative angiographic analysis.
Marie-Josée Cloutier+4 more
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Pathophysiology of coronary artery in‐stent restenosis
Acute Cardiac Care, 2007In-stent restenosis reflects the interaction of a cascade of molecular and cellular events occurring within the vessel wall. Coronary stenting induces localized injury to the vessel wall, which leads to the release of thrombogenic, vasoactive, and lymphocytes mitogenic factors that result in processes causing re-narrowing at the injured site.
A. Kibos, I. Tintoiu, A. Campeanu
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Restenosis After Coronary Balloon Angioplasty
Annual Review of Medicine, 1991A recurrence of stenosis (restenosis) following successful coronary angioplasty continues to be a frequent problem limiting the long-term efficacy of the procedure. An overexuberant reparative response to the arterial injury induced by balloon dilatation leads to intimal hyperplasia, the major mechanism responsible for restenosis. Although none has yet
Richard A. Lange+2 more
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Trends and Outcomes of Restenosis After Coronary Stent Implantation in the United States.
Journal of the American College of Cardiology, 2020I. Moussa+9 more
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