Results 211 to 220 of about 544,583 (258)
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Familial Recurrent Cranial Nerve Palsies
Acta Neurologica Scandinavica, 1988Family cases of recurrent cranial nerve palsies are seldom reported. This paper presents a family with recurrent facial and ocular nerve palsies in 2 brothers. Their father and his sister had Bell's palsies. Examinations provided no explanation. Six previous reports of families with recurrent cranial nerve palsies are summarized. The pedigrees speak in
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Pediatric Cranial Nerve Palsies
International Ophthalmology ClinicsCranial nerve palsies in children offer unique challenges distinct from those in adults, and typically arise from congenital, traumatic, neoplastic, or postinfectious inflammatory disease. With rare reports of diplopia, diagnosis depends on indirect signs such as abnormal head posture, strabismus, or abnormal gaze. The oculomotor (III), trochlear (IV),
Michael Nguyen +3 more
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Sensory ataxia with cranial nerve palsies
Practical Neurology, 2021A previously independent 64-year-old man presented to his local hospital with new-onset diplopia and dysarthria while watching television. Over the following days, he developed painless perioral and upper limb sensory disturbance, gait unsteadiness and limb ataxia, most pronounced in the upper limbs.
Alexander, Grundmann +3 more
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2013
This chapter describes the neuroanatomy and common lower cranial neuropathies, including clinical presentation, evaluation, diagnosis, and treatment of the following cranial nerves: trigeminal (fifth), facial (seventh), glossopharyngeal (ninth), vagus (tenth), spinal accessory (eleventh), and hypoglossal (twelfth).
Vita Grynova Kesner, Christina Fournier
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This chapter describes the neuroanatomy and common lower cranial neuropathies, including clinical presentation, evaluation, diagnosis, and treatment of the following cranial nerves: trigeminal (fifth), facial (seventh), glossopharyngeal (ninth), vagus (tenth), spinal accessory (eleventh), and hypoglossal (twelfth).
Vita Grynova Kesner, Christina Fournier
openaire +1 more source
2015
The clinical features of superior oblique paresis vary widely depending on the type. Common features include an ipsilateral hypertropia that increases on contralateral gaze, and a positive head tilt test with the hypertropia increasing on head tilt to the side of the hypertropia.
Kenneth W. Wright, Yi Ning J. Strube
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The clinical features of superior oblique paresis vary widely depending on the type. Common features include an ipsilateral hypertropia that increases on contralateral gaze, and a positive head tilt test with the hypertropia increasing on head tilt to the side of the hypertropia.
Kenneth W. Wright, Yi Ning J. Strube
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Third cranial nerve palsy in children
American Journal of Ophthalmology, 1999To report the causes and the sensory, motor, and cosmetic results after treatment for oculomotor (third cranial nerve) palsy in children.Review of the clinical records of children with a diagnosis of third cranial nerve palsy followed up in a university-based pediatric ophthalmology practice between 1981 and 1996.Forty-nine children with 53 affected ...
L A, Schumacher-Feero +3 more
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Slowly progressive cranial nerve palsies
Medical Journal of Australia, 2006Perineural spread (PNS) from these lesionsinvolves either small nerves, identified at pathological examina-tion (incidental), or large nerves, presenting clinically as cranialnerve palsies. Basal cell carcinoma is the more common skincancer, but incidental PNS is most frequently associated withsquamous cell carcinoma.
Warner, Giles C. +2 more
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