A study of creep of titanium and two of its alloys : fourth progress report. [PDF]
Jeremy V. Gluck, James W. Freeman
openalex
Discussion: “Progress Report on Tubular Creep Tests” (Norton, F. H., 1941, Trans. ASME, 63, pp. 735–736) and “Interpretation of Creep Tests on Tubes” (Soderberg, C. R., 1941, Trans. ASME, 63, pp. 737–740) [PDF]
Ernest L. Robinson
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Targeted protein degradation in oncology: novel therapeutic opportunity for solid tumours?
Current anticancer therapies are limited by the occurrence of resistance and undruggability of most proteins. Targeted protein degraders are novel, promising agents that trigger the selective degradation of previously undruggable proteins through the recruitment of the ubiquitin–proteasome machinery. Their mechanism of action raises exciting challenges,
Noé Herbel, Sophie Postel‐Vinay
wiley +1 more source
Vascular endothelial‐cadherin as a marker of endothelial injury in preclinical Alzheimer disease
Abstract Objective Endothelial dysfunction is an early and prevalent pathology in Alzheimer disease (AD). We here investigate the value of vascular endothelial‐cadherin (VEC) as a cerebrospinal fluid (CSF) marker of endothelial injury in preclinical AD.
Rawan Tarawneh+5 more
wiley +1 more source
Understanding the creep behaviors and mechanisms of Mg-Gd-Zn alloys via machine learning
Mg-Gd-Zn based alloys have better creep resistance than other Mg alloys and attract more attention at elevated temperatures. However, the multiple alloying elements and various heat treatment conditions, combined with complex microstructural evolution ...
Shuxia Ouyang+9 more
doaj
Study on creep hardening-damage constitutive model of cemented tailings backfill based on time-harden theory. [PDF]
Feng Y, Sun G, Liu Z.
europepmc +1 more source
Discussion: “The Interpretation of Creep Tests for Machine Design” (Soderberg, C. Richard, 1936, Trans. ASME, 58, pp. 733–743) [PDF]
Joseph Marin
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Modeling of Creep Deformation and Creep Fracture
Qiang Xu, Zhongyu Lu
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Nuclear prothymosin α inhibits epithelial‐mesenchymal transition (EMT) in lung cancer by increasing Smad7 acetylation and competing with Smad2 for binding to SNAI1, TWIST1, and ZEB1 promoters. In early‐stage cancer, ProT suppresses TGF‐β‐induced EMT, while its loss in the nucleus in late‐stage cancer leads to enhanced EMT and poor prognosis.
Liyun Chen+12 more
wiley +1 more source