Results 101 to 110 of about 795,041 (278)

STUDIES ON BLOOD GLUCOSE, TOTAL PROTEINS, UREA AND CHOLESTEROL LEVELS IN CYCLIC, NON-CYCLIC AND ENDOMETRITIC CROSSBRED COWS [PDF]

open access: yesPakistan Veterinary Journal, 2004
Seventy-five crossbred cows kept at the Livestock Experimental Station, Qadirabad, District Sahiwal were divided into three equal groups i.e. cyclic, non cyclic and endometritic.
Ijaz Ahmad, L.A. Lodhi, Z.I. Qureshi and M. Younis1
doaj  

Entropies of Weighted Sums in Cyclic Groups and an Application to Polar Codes

open access: yesEntropy, 2017
In this note, the following basic question is explored: in a cyclic group, how are the Shannon entropies of the sum and difference of i.i.d. random variables related to each other?
Emmanuel Abbe   +2 more
doaj   +1 more source

A synthetic benzoxazine dimer derivative targets c‐Myc to inhibit colorectal cancer progression

open access: yesMolecular Oncology, EarlyView.
Benzoxazine dimer derivatives bind to the bHLH‐LZ region of c‐Myc, disrupting c‐Myc/MAX complexes, which are evaluated from SAR analysis. This increases ubiquitination and reduces cellular c‐Myc. Impairing DNA repair mechanisms is shown through proteomic analysis.
Nicharat Sriratanasak   +8 more
wiley   +1 more source

Almost cyclic groups

open access: yes, 2005
A group G is almost cyclic if there is an element x in G, such that for all g in G, there is an element y in G and an integer n with ygy^{-1} = x^n (that is, every element is conjugate to some power of x). W. Ziller asked whether there are finitely-presented almost cyclic groups which are not cyclic in connection with work on closed geodesics. V.
openaire   +2 more sources

Patient‐specific pharmacogenomics demonstrates xCT as predictive therapeutic target in colon cancer with possible implications in tumor connectivity

open access: yesMolecular Oncology, EarlyView.
This study integrates transcriptomic profiling of matched tumor and healthy tissues from 32 colorectal cancer patients with functional validation in patient‐derived organoids, revealing dysregulated metabolic programs driven by overexpressed xCT (SLC7A11) and SLC3A2, identifying an oncogenic cystine/glutamate transporter signature linked to ...
Marco Strecker   +16 more
wiley   +1 more source

Aggressive prostate cancer is associated with pericyte dysfunction

open access: yesMolecular Oncology, EarlyView.
Tumor‐produced TGF‐β drives pericyte dysfunction in prostate cancer. This dysfunction is characterized by downregulation of some canonical pericyte markers (i.e., DES, CSPG4, and ACTA2) while maintaining the expression of others (i.e., PDGFRB, NOTCH3, and RGS5).
Anabel Martinez‐Romero   +11 more
wiley   +1 more source

Glycosylated LGALS3BP is highly secreted by bladder cancer cells and represents a novel urinary disease biomarker

open access: yesMolecular Oncology, EarlyView.
Urinary LGALS3BP is elevated in bladder cancer patients compared to healthy controls as detected by the 1959 antibody–based ELISA. The antibody shows enhanced reactivity to the high‐mannose glycosylated variant secreted by cancer cells treated with kifunensine (KIF).
Asia Pece   +18 more
wiley   +1 more source

Cyclic Quotients of Transitive Groups

open access: yesJournal of Algebra, 2000
In this note two interesting theorems about finite permutation groups and an application to the Galois theory of function fields resp. algebraic curves are proved. Theorem 1. If \(A\) is a transitive subgroup of the symmetric group \(S_n\) on \(n\) letters and \(G\) is a normal subgroup of \(A\) such that \(A/G\) is cyclic, then \(|A/G|\leq n\) and ...
openaire   +2 more sources

Survivin and Aurora Kinase A control cell fate decisions during mitosis

open access: yesMolecular Oncology, EarlyView.
Aurora A interacts with survivin during mitosis and regulates its centromeric role. Loss of Aurora A activity mislocalises survivin, the CPC and BubR1, leading to disruption of the spindle checkpoint and triggering premature mitotic exit, which we refer to as ‘mitotic slippage’.
Hana Abdelkabir   +2 more
wiley   +1 more source

CDK11 inhibition induces cytoplasmic p21WAF1 splice variant by p53 stabilisation and SF3B1 inactivation

open access: yesMolecular Oncology, EarlyView.
CDK11 inhibition stabilises the tumour suppressor p53 and triggers the production of an alternative p21WAF1 splice variant p21L, through the inactivation of the spliceosomal protein SF3B1. Unlike the canonical p21WAF1 protein, p21L is localised in the cytoplasm and has reduced cell cycle‐blocking activity.
Radovan Krejcir   +12 more
wiley   +1 more source

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