Results 161 to 170 of about 194,175 (208)

POSTCATARACT CYSTOID MACULAR EDEMA

Ophthalmology Clinics of North America, 2001
Ocular tissues, like those of other organs, exhibit well-defined morphologic reactions to local trauma and insult in the form of hyperemia, vasodilation, increased permeability of blood vessels, and edema. Cystoid macular edema (CME) following cataract surgery is one such manifestation, and it can result in either temporary, or rarely, a permanent ...
M, Nagpal, K, Nagpal, P N, Nagpal
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Pseudophakic cystoid macular edema

Seminars in Ophthalmology, 2002
Cystoid macular edema (CME) following cataract surgery has been recognized for over 50 years as an important cause of suboptimal post-operative vision. The incidence of CME varies widely, but is likely in the range of 1-2% using modern cataract extraction techniques.
Subhransu, Ray, Donald J, D'Amico
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Pseudophakic cystoid macular edema

Current Opinion in Ophthalmology, 2012
Pseudophakic cystoid macular edema (PCME) is a common cause of visual impairment after cataract surgery. This article systematically reviews and discusses the epidemiology, risk factors, diagnosis, and treatment of PCME, with a focus on advances in the past 1-2 years.The incidence of PCME has declined with the advent of modern surgical techniques ...
Yoshihiro, Yonekawa, Ivana K, Kim
openaire   +2 more sources

Inflammatory cystoid macular edema

Current Opinion in Ophthalmology, 2007
The aim of this article is to update our current understanding and management of inflammatory cystoid macular edema.Cystoid macular edema is a common cause of visual loss in uveitis, which occurs predominantly in older patients with chronic uveitis forms and might be heralded by subclinical changes on optic coherence tomography.
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Cystoid macular edema in uveitis

Ocular Immunology and Inflammation, 2003
Cystoid macular edema (CME) can cause profound visual loss and is one of the major causes of legal blindness in patients with uveitis. It can complicate virtually any type of acute or chronic, anterior or posterior uveitis. When mild and of short duration, CME may respond to treatment used to control the intraocular inflammation.
Narciss, Okhravi, Susan, Lightman
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Vitrectomy and Cystoid Macular Edema

Ophthalmology, 1980
Twenty-two patients who underwent vitrectomy surgery for chronic cystoid macular edema (CME) were evaluated. These patients all had uneventful intracapsular cataract extraction, but CME with vitreous adhesions to the cataract incision later developed.
J L, Federman, W H, Annesley, L K, Sarin, P, Remer   +3 more
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Cystoid Macular Edema in Uveitis

2010
Macular edema is a major cause of morbidity in uveitis patients. Inflammatory mediators act on the integrity of the blood ocular barrier and on the function of the RPE pump. Chronicity leads to irreversible changes and is reported to cause up to 30% of permanent visual loss in uveitis patients.
Marc D, de Smet, Annabelle A, Okada
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Vitrectomy for Diabetic Cystoid Macular Edema

Japanese Journal of Ophthalmology, 2001
We evaluated the visual outcome of vitrectomy for diabetic cystoid macular edema.Visual outcome and factors which may influence final visual acuity were assessed and documented retrospectively in 45 eyes of 40 patients, all of whom were followed for at least 6 months postoperatively.Final postoperative visual acuity was two or more lines better than ...
Yukihiro, Sato, Zeon, Lee, Hiroyuki, Shimada   +2 more
openaire   +3 more sources

Pathology of Cystoid Macular Edema

Ophthalmology, 1982
In order to define the pathology of cystoid macular edema (CME), 35 human cases with histopathologic features of CME were reviewed. The macular cysts appeared in different layers of the retina in eyes after cataract extraction, retinal vein occlusion, trauma, diabetes mellitus, and accelerated hypertension.
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Pseudophakic cystoid macular edema

Current Opinion in Ophthalmology
Purpose of review Pseudophakic cystoid macular edema (PCME) is the most common postoperative complication of cataract surgery, resulting in visual decline. In this review, we discuss its pathophysiology, epidemiology, clinical presentation, and the current available evidence on therapeutic management.
Yousef A, Fouad, Sam, Karimaghaei, Abdelrahman M, Elhusseiny, Ahmed R, Alagorie, Andrew D, Brown, Ahmed B, Sallam   +5 more
openaire   +2 more sources